EZHS42
Human Amyloid β42 ELISA Kit
measures and quantifies Amyloid β42 levels in 50 μL CSF, cell culture supernatent or plasma
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96 WELLS
4290,00 zł
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96 WELLS
4290,00 zł
About This Item
Kod UNSPSC:
12161503
eCl@ss:
32161000
NACRES:
NA.84
Polecane produkty
Nazwa produktu
High Sensitivity Human Amyloid β42 ELISA, This High Sensitivity Human Amyloid β42 ELISA is used to measure & quantify Amyloid β42 levels in Neuroscience research.
Poziom jakości
reaktywność gatunkowa
human
opakowanie
kit of 1 × 96 wells
Parametry
50 μL sample volume (Overnight assay)
assay range
sensitivity: 8.0 pg/mL
(50 μl sample size)
standard curve range: 16-500 pg/mL
metody
ELISA: suitable
moc wejściowa
sample type plasma (K2 EDTA)
sample type serum
sample type cerebrospinal fluid (CSF)
Zastosowanie
research use
metoda wykrywania
colorimetric (450nm/590nm)
Warunki transportu
wet ice
temp. przechowywania
2-8°C
informacje o genach
human ... APP(351)
Opis ogólny
Amyloid beta peptides have been implicated in the etiology of Alzheimer’s disease. Amyloid beta 40 is the most prominent peptide and Amyloid beta 42 is the neurotoxic form. The Amyloid beta 42/40-ratio (AB ratio) has been reported as a better indicator of the Alzheimer pathology. Millipore’s High Sensitivity Human Amyloid β42 ELISA kit is used for the measurement of Amyloid β42 in cerebrospinal fluid, cell culture supernatants, primary neurons and plasma in a 96-well format.
Specyficzność
The Amyloid β42 ELISA (HS) uses monoclonal anti-Aβ antibodies with high selectivity for human Aβ. The capture antibody recognizes the C-terminal end of Amyloid β1-42, which causes a high selectivity for Aβ42. The cross-reactivity of the used antibodies to other Amyloid peptides was tested by ELISA and BIACORE and shows no significant cross-reactivity to Aβ1-38, Aβ1-39, Aβ1-40, Aβ1-43 and Aβ1-44.
Zastosowanie
Research Category
Neuroscience
Neuroscience
Research Sub Category
Alzheimer′s Disease
Alzheimer′s Disease
This High Sensitivity Human Amyloid β42 ELISA is used to measure & quantify Amyloid β42 levels in Neuroscience research.
This assay requires 50 µl of sample and is an overnight assay.
Used to detect/quantify: Amyloid β42
Przechowywanie i stabilność
Components in the kit can be stored up to 2 weeks at 2-8°C
Inne uwagi
Please contact Technical Service for linearity of dilution.
Oświadczenie o zrzeczeniu się odpowiedzialności
For research use only. Not for use in diagnostic procedures.
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Hasło ostrzegawcze
Warning
Zwroty wskazujące rodzaj zagrożenia
Zwroty wskazujące środki ostrożności
Klasyfikacja zagrożeń
Aquatic Chronic 3 - Met. Corr. 1 - Skin Sens. 1
Kod klasy składowania
8A - Combustible corrosive hazardous materials
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Dokumenty związane z niedawno zakupionymi produktami zostały zamieszczone w Bibliotece dokumentów.
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Current biology : CB, 26(17), 2291-2300 (2016-08-16)
Glucose hypometabolism is a prominent feature of the brains of patients with Alzheimer's disease (AD). Disease progression is associated with a reduction in glucose transporters in both neurons and endothelial cells of the blood-brain barrier. However, whether increasing glucose transport
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Psychiatry investigation, 15(2), 205-213 (2018-02-25)
Conventional methods for organotypic hippocampal tissue slice culture (OHSC) have shown several disadvantages or limitations regarding age of animals used, duration of culture and difficulty using neurodegenerative models. Therefore, we tried to establish OHSC from old 3xTg-Alzheimer's disease (AD) mice
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Acta myologica : myopathies and cardiomyopathies : official journal of the Mediterranean Society of Myology, 33(1), 13-18 (2014-05-21)
The presence of non-progressive cognitive impairment is recognized as a common feature in a substantial proportion of patients with Duchenne muscular dystrophy (DMD). Concurrently, the amyloid beta peptide (Aβ42) protein has been associated with changes in memory and cognitive functions.
Nataliya Golovyashkina et al.
Molecular neurodegeneration, 10, 60-60 (2015-11-07)
Dendritic simplification, a key feature of the neurodegenerative triad of Alzheimer's disease (AD) in addition to spine changes and neuron loss, occurs in a region-specific manner. However, it is unknown how changes in dendritic complexity are mediated and how they
Gunjan Manocha et al.
Current Alzheimer research, 15(12), 1123-1135 (2018-08-03)
Alzheimer's disease (AD) is associated with age-associated central nervous system degeneration and dementia. This decline in the function correlates with deposition of Aβ peptide containing plaques and associated reactive gliosis. The inflammatory phenotype of microglia, in particular, is often considered
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