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H145
(±)-7-hydroxy-dpat hydrobromide
Synonym(e):
(±)-2-Dipropylamino-7-hydroxy-1,2,3,4-tetrahydronaphthalene hydrobromide
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About This Item
Empirische Formel (Hill-System):
C16H25NO · HBr
CAS-Nummer:
Molekulargewicht:
328.29
MDL-Nummer:
UNSPSC-Code:
12352200
SMILES String
Br.CCCN(CCC)C1CCc2ccc(O)cc2C1
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Produkt-Nr.
Beschreibung
Preisangaben
Lagerklassenschlüssel
13 - Non Combustible Solids
WGK
WGK 3
Flammpunkt (°F)
Not applicable
Flammpunkt (°C)
Not applicable
Persönliche Schutzausrüstung
Eyeshields, Gloves, type N95 (US)
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Eriko Ikeda et al.
Molecular pharmacology, 83(5), 959-967 (2013-02-23)
The dopamine D3 receptor (DRD3) in the ventral striatum is thought to influence motivation and motor functions. Although the expression of DRD3 in the ventral striatum has been shown to exhibit 24-hour variations, the mechanisms underlying the variation remain obscure.
Mikhail N Koffarnus et al.
Psychopharmacology, 203(2), 317-327 (2008-09-23)
Previous research has found the stimulus effects of dopamine D2- and D3-preferring agonists difficult to distinguish in drug discrimination studies. Antagonism studies suggest that the stimulus effects of both types of agonists may be mediated primarily through D2 receptors. The
Gavin D Phillips et al.
Psychopharmacology, 203(1), 161-173 (2008-10-25)
Two issues were addressed regarding the effects of amygdala dopamine manipulations on associative learning: first, an apparent contradiction between the effects of post- vs. pre-session dopaminergic manipulations and second, the ability of dopaminergic infusions to affect association formation vs. its
Rosario Sánchez-Pernaute et al.
Neurobiology of disease, 27(2), 220-227 (2007-06-26)
A growing body of evidence indicates a role for D(3) receptors in l-DOPA-induced dyskinesias. This involvement could be amenable to non-invasive in vivo analysis using functional neuroimaging. With this goal, we examined the hemodynamic response to the dopamine D(3)-preferring agonist
Kenneth J Thiel et al.
Behavioural brain research, 214(2), 386-394 (2010-07-06)
Alterations in dopamine output within the various subnuclei of the amygdala have previously been implicated in cocaine reinforcement, as well as cocaine-seeking behavior. To elucidate the potential for increased stimulation of D1- and D2-like receptors (D1Rs and D2Rs, respectively) specifically
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