07-689
Anti-EZH2-Antikörper
Upstate®, from rabbit
Synonym(e):
KMT6, Enx1, Enhancer of Zeste Homologue 2
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| Packungsgröße | SKU | Verfügbarkeit | Preis |
|---|
Über diesen Artikel
biological source
rabbit
Quality Level
conjugate
unconjugated
antibody form
whole antiserum
antibody product type
primary antibodies
clone
polyclonal
species reactivity
human, mouse
packaging
antibody small pack of 25 μL
manufacturer/tradename
Upstate®
technique(s)
ChIP: suitable (ChIP-seq), immunoprecipitation (IP): suitable, western blot: suitable
NCBI accession no.
UniProt accession no.
shipped in
ambient
target post-translational modification
unmodified
Gene Information
human ... EZH2(2146)
General description
Immunogen
Application
Histone
Epigenetik & Zellkernfunktion
Biochem/physiol Actions
Physical form
Preparation Note
Analysis Note
K562-Zelllysat, C2C12-Zelllysat
Other Notes
Legal Information
Disclaimer
1 of 1
Dieser Artikel | |||
|---|---|---|---|
| antibody form whole antiserum | antibody form serum | antibody form affinity isolated antibody | antibody form purified immunoglobulin |
| biological source rabbit | biological source rabbit | biological source rabbit | biological source mouse |
| species reactivity human, mouse | species reactivity rat, mouse, human | species reactivity human, mouse | species reactivity human, mouse |
| conjugate unconjugated | conjugate unconjugated | conjugate unconjugated | conjugate unconjugated |
| clone polyclonal | clone polyclonal | clone polyclonal | clone BD43, monoclonal |
| shipped in ambient | shipped in wet ice | shipped in wet ice | shipped in wet ice |
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Lagerklasse
12 - Non Combustible Liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
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Verwandter Inhalt
Cancer is a complex disease manifestation. At its core, it remains a disease of abnormal cellular proliferation and inappropriate gene expression. In the early days, carcinogenesis was viewed simply as resulting from a collection of genetic mutations that altered the gene expression of key oncogenic genes or tumor suppressor genes leading to uncontrolled growth and disease (Virani, S et al 2012). Today, however, research is showing that carcinogenesis results from the successive accumulation of heritable genetic and epigenetic changes. Moreover, the success in how we predict, treat and overcome cancer will likely involve not only understanding the consequences of direct genetic changes that can cause cancer, but also how the epigenetic and environmental changes cause cancer (Johnson C et al 2015; Waldmann T et al 2013). Epigenetics is the study of heritable gene expression as it relates to changes in DNA structure that are not tied to changes in DNA sequence but, instead, are tied to how the nucleic acid material is read or processed via the myriad of protein-protein, protein-nucleic acid, and nucleic acid-nucleic acid interactions that ultimately manifest themselves into a specific expression phenotype (Ngai SC et al 2012, Johnson C et al 2015). This review will discuss some of the principal aspects of epigenetic research and how they relate to our current understanding of carcinogenesis. Because epigenetics affects phenotype and changes in epigenetics are thought to be key to environmental adaptability and thus may in fact be reversed or manipulated, understanding the integration of experimental and epidemiologic science surrounding cancer and its many manifestations should lead to more effective cancer prognostics as well as treatments (Virani S et al 2012).
Global Trade Item Number
| SKU | GTIN |
|---|---|
| 07-689-25UL | 04054839350474 |