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Merck
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重要文件

57697

Sigma-Aldrich

Advanced Protein Assay

1× concentrate

同義詞:

Protein Assay, advanced

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About This Item

分類程式碼代碼:
12171500
NACRES:
NA.51
暫時無法取得訂價和供貨情況

儲存溫度

2-8°C

一般說明

The Advanced Protein Assay Reagent combines the best properties of low protein-to-protein variance and a wide protein concentration, a sensitive detection limit and a unique red-to-blue color change.

訊號詞

Warning

危險聲明

危險分類

Acute Tox. 4 Oral - Met. Corr. 1 - STOT SE 2

標靶器官

Eyes,Central nervous system

儲存類別代碼

8A - Combustible corrosive hazardous materials

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter


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Mathieu Panel et al.
The Journal of pharmacology and experimental therapeutics, 376(3), 348-357 (2020-12-12)
Mitochondrial permeability transition pore (mPTP) opening is a key event in cell death during myocardial ischemia reperfusion. Inhibition of its modulator cyclophilin D (CypD) by cyclosporine A (CsA) reduces ischemia-reperfusion injury. The use of cyclosporine A in this indication is
Romain Long et al.
Biochimica et biophysica acta, 1837(1), 201-209 (2013-10-29)
Carbon monoxide is continuously produced in small quantities in tissues and is an important signaling mediator in mammalian cells. We previously demonstrated that CO delivered to isolated rat heart mitochondria using a water-soluble CO-releasing molecule (CORM-3) is able to uncouple
Stéphanie Paradis et al.
Cardiovascular research, 98(3), 420-427 (2013-04-05)
The translocator protein (TSPO) is located on the outer mitochondrial membrane where it is responsible for the uptake of cholesterol into mitochondria of steroidogenic organs. TSPO is also present in the heart where its role remains uncertain. We recently showed
Mathieu Panel et al.
Scientific reports, 7(1), 4283-4283 (2017-06-29)
Opening of the mitochondrial permeability transition pore (mPTP) plays a major role in cell death during cardiac ischaemia-reperfusion. Adult isolated rodent cardiomyocytes are valuable cells to study the effect of drugs targeting mPTP. This study investigated whether the use of
Matthias Kohlhauer et al.
Shock (Augusta, Ga.), 56(5), 857-864 (2021-05-13)
Mitochondria is often considered as the common nexus of cardiac and cerebral dysfunction after cardiac arrest. Here, our goal was to determine whether the time course of cardiac and cerebral mitochondrial dysfunction is similar after shockable versus non-shockable cardiac arrest

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