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03553

Supelco

Griess′ reagent for nitrite

for microscopy

Synonim(y):

Griess-Ilosvays Reagent for nitrate, Lunge’s Reagent

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About This Item

Numer MDL:
MFCD01866819
Kod UNSPSC:
12164500
NACRES:
NA.21

klasa czystości

for microscopy

Postać

liquid

zanieczyszczenia

2-naphthylamine
 acetic acid

współczynnik refrakcji

n20/D 1.353

gęstość

1.037 g/mL at 20 °C

Zastosowanie

food and beverages
general analytical

Opis ogólny

The Griess reagent is used for the quantitative analysis of nitrite ions. Present nitrite (as well as nitrate) ions will form a pink diazo dye by diazonium coupling reaction with N-(1-Naphthyl)ethylenediamine. The concentration of nitrite (or nitrate) ions is proportional to the color intensity of the resulting red solution.
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Piktogramy

Health hazardCorrosion
GHS08,GHS05

Hasło ostrzegawcze

Danger

Zwroty wskazujące rodzaj zagrożenia

H314,H350

Klasyfikacja zagrożeń

Carc. 1A - Eye Dam. 1 - Skin Corr. 1B

Kod klasy składowania

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

Klasa zagrożenia wodnego (WGK)

WGK 3

Temperatura zapłonu (°F)

Not applicable

Temperatura zapłonu (°C)

Not applicable

Środki ochrony indywidualnej

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter

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Certyfikaty analizy (CoA)

Lot/Batch Number
BCCB9005
BCBZ2918
BCBT1248
BCBT1571
BCBQ4791V

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Dokumenty związane z niedawno zakupionymi produktami zostały zamieszczone w Bibliotece dokumentów.

Odwiedź Bibliotekę dokumentów

Su Young Chae et al.
Biomaterials, 25(5), 843-850 (2003-11-12)
Pancreatic islets and insulinoma cells are particularly vulnerable to serious damage by cytotoxic nitric oxide (NO) and/or oxidative stress, most probably due to their low expression levels of antioxidant enzymes. This cellular damage has been regarded as one of major
Anne M Sanders et al.
BMC cancer, 10, 121-121 (2010-04-02)
Spontaneous Regression/Complete Resistant (SR/CR) mice are a colony of cancer-resistant mice that can detect and rapidly destroy malignant cells with innate cellular immunity, predominately mediated by granulocytes. Our previous studies suggest that several effector mechanisms, such as perforin, granzymes, or
A Campos-Neto et al.
Journal of immunology (Baltimore, Md. : 1950), 160(5), 2037-2041 (1998-03-14)
It has been proposed that the induction of cellular immunity and resistance to intracellular pathogens is dependent upon CD40 ligand (CD40L). In the present study we show that this proposal is not ubiquitously supported. Mice genetically deficient in CD40L (CD40LKO)
Qing-Li Wu et al.
Journal of immunology (Baltimore, Md. : 1950), 182(11), 6771-6778 (2009-05-21)
The Emu-TCL1 transgenic mouse spontaneously develops a CD5(+) B cell lymphoproliferative disorder similar to human chronic lymphocytic leukemia (CLL). Given the ineffectual T cell antitumor responses in this mouse model of CLL, we sought to determine whether combined treatment with
Lei Qiu et al.
Immunology, 128(1 Suppl), e325-e334 (2008-11-20)
Using two-dimensional sodium dodecyl sulphate-polyacrylamide gel electrophoresis, we found that copper/zinc superoxide dismutase (Cu/Zn-SOD, SOD-1) was induced in constructed CCR5 stably transfected HEK 293 cells, but not in mock cells, treated with CCL5. CCL5-induced SOD-1 expression was also confirmed in

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