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SAB1305552

Sigma-Aldrich

MONOCLONAL ANTI-LC3 (APG8) antibody produced in mouse

clone 166AT1234, IgG fraction of antiserum, buffered aqueous solution

Synonym(e):

Autophagy-related protein LC3 A, Autophagy-related ubiquitin-like modifier LC3 A, MAP1 light chain 3-like protein 1, MAP1LC3A, Microtubule-associated proteins 1A/1B light chain 3A

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About This Item

UNSPSC-Code:
12352203
NACRES:
NA.41

Biologische Quelle

mouse

Qualitätsniveau

100

Antikörperform

IgG fraction of antiserum

Antikörper-Produkttyp

primary antibodies

Klon

166AT1234, monoclonal

Form

buffered aqueous solution

Mol-Gew.

14272 Da

Speziesreaktivität

rat, mouse, human

Methode(n)

immunofluorescence: 1:25
immunohistochemistry: 1:50-1:100
western blot: 1:1000

Isotyp

IgG1κ

NCBI-Hinterlegungsnummer

NP_115903.1

UniProt-Hinterlegungsnummer

Q9H492

Versandbedingung

wet ice

Lagertemp.

−20°C

Posttranslationale Modifikation Target

unmodified

Angaben zum Gen

human ... MAP1LC3A(84557)

Physikalische Form

Supplied in PBS with 0.09% (W/V) sodium azide

Haftungsausschluss

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Lagerklassenschlüssel

10 - Combustible liquids

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Xiaoming Shu et al.
Molecular medicine reports, 16(2), 1180-1188 (2017-06-07)
Peripheral blood T lymphocytopenia has previously been identified in polymyositis/dermatomyositis (PM/DM) patients. Therefore, the present study aimed to examine the potential role of autophagy in peripheral blood T cell survival in PM/DM patients. Transmission electron microscopy was used to detect
Matteo Bordoni et al.
Cells, 11(8) (2022-04-24)
Mitochondria alterations are present in tissues derived from patients and animal models, but no data are available for peripheral blood mononuclear cells (PBMCs) of ALS patients. This work aims to investigate mitophagy in PBMCs of sporadic (sALS) patients and how
Hong Ding et al.
Journal of asthma and allergy, 17, 717-731 (2024-08-06)
Accumulating evidence indicates that oxidative stress and inflammation are the pathological basis of allergic diseases. Inhibition of NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome could ameliorate allergic rhinitis (AR). Here, we explored the effects and mechanisms that underlie NLRP3
Maomao Sun et al.
Frontiers in immunology, 12, 685523-685523 (2021-08-03)
Recent studies have shown that autophagy upregulation can attenuate sepsis-induced acute kidney injury (SAKI). The tumor suppressor p53 has emerged as an autophagy regulator in various forms of acute kidney injury (AKI). Our previous studies showed that p53 acetylation exacerbated
Rongrong Hua et al.
Journal of cellular biochemistry, 120(9), 15915-15923 (2019-05-14)
The sequential reactivation of mechanistic target of rapamycin (mTOR) inhibited autophagic flux in neurons exposed to oxygen-glucose deprivation/reperfusion (OGD/R), which was characterized by reduction of autophagosome formation and restriction of autolysosome degradation. However, its detailed molecular mechanism was still unknown.
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