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203122

Sigma-Aldrich

Kupfer

powder, 99.99% trace metals basis

Synonym(e):

Cu powder

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About This Item

Empirische Formel (Hill-System):
Cu
CAS-Nummer:
7440-50-8
Molekulargewicht:
63.55
EG-Nummer:
231-159-6
MDL-Nummer:
MFCD00010965
UNSPSC-Code:
12141711
eCl@ss:
38150101
PubChem Substanz-ID:
24852148
NACRES:
NA.23

Qualitätsniveau

100

Assay

99.99% trace metals basis

Form

powder

Widerstandsfähigkeit

1.673 μΩ-cm, 20°C

bp

2567 °C (lit.)

mp (Schmelzpunkt)

1083.4 °C (lit.)

Dichte

8.94 g/mL at 25 °C (lit.)

SMILES String

[Cu]

InChI

1S/Cu

InChIKey

RYGMFSIKBFXOCR-UHFFFAOYSA-N

Allgemeine Beschreibung

Copper powder, 99.999% trace metals basis, is a fine powder that is typically red-brown in color. It is often produced through the reduction of copper oxide or other copper compounds using hydrogen or other reducing agents. Our powder is highly pure, with less than 15 ppm impurities such as lead, arsenic, and bismuth, making it suitable for a wide range of industrial and commercial applications. Copper is valued for its excellent electrical and thermal conductivity, high ductility, and resistance to corrosion.

Anwendung

High-purity copper powder is used in a variety of applications, such as in the production of electrical components and conductive coatings and as a catalyst in chemical reactions. Additionally, copper powder can be used in the production of various alloys and as a raw material for powder metallurgy.

Leistungsmerkmale und Vorteile

Our 99.999% pure copper powder is useful in the electronics and chemical industries where purity matters most.
✔ Consistent quality
✔ Ultra high purity
✔ Available to scale up: bulk and pilot scale

Signalwort

Danger-Danger

Gefahreneinstufungen

Aquatic Acute 1 - Aquatic Chronic 1 - Flam. Sol. 1

Lagerklassenschlüssel

4.1B - Flammable solid hazardous materials

WGK

WGK 2

Flammpunkt (°F)

212.0 °F

Flammpunkt (°C)

100 °C

Persönliche Schutzausrüstung

Eyeshields, Gloves, type P3 (EN 143) respirator cartridges

Hier finden Sie alle aktuellen Versionen:

Analysenzertifikate (COA)

Lot/Batch Number
MKCW6084
MKCN0269
MKCL3008
MKCG3837
MKCD8741

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Kupfer fine powder particle size 230 mesh ASTM) EMSURE®

Supelco

1.02703

Kupfer

Kupfer beads, 2-8 mm, ≥99.99% trace metals basis

Sigma-Aldrich

326488

Kupfer

Kupfer nanopowder, 25 nm particle size (TEM)

Sigma-Aldrich

774081

Kupfer

Niobium carbide powder, 45 max. part. size (micron), weight 50 g, purity 99%

GF66605788

Niobium carbide

Stephen G Kaler et al.
The New England journal of medicine, 358(6), 605-614 (2008-02-08)
Menkes disease is a fatal neurodegenerative disorder of infancy caused by diverse mutations in a copper-transport gene, ATP7A. Early treatment with copper injections may prevent death and illness, but presymptomatic detection is hindered by the inadequate sensitivity and specificity of
Daniel L Priebbenow et al.
Organic letters, 15(24), 6155-6157 (2013-11-28)
A method has been developed for the preparation of N-alkynylated sulfoximines involving the copper-catalyzed decarboxylative coupling of sulfoximines with aryl propiolic acids. A range of substituents on both the sulfoximidoyl moiety and the aryl group of the propiolic acid were
Hiroshi Sato et al.
Science (New York, N.Y.), 343(6167), 167-170 (2013-12-18)
Carbon monoxide (CO) produced in many large-scale industrial oxidation processes is difficult to separate from nitrogen (N2), and afterward, CO is further oxidized to carbon dioxide. Here, we report a soft nanoporous crystalline material that selectively adsorbs CO with adaptable
Magnus Andersson et al.
Nature structural & molecular biology, 21(1), 43-48 (2013-12-10)
Heavy metals in cells are typically regulated by PIB-type ATPases. The first structure of the class, a Cu(+)-ATPase from Legionella pneumophila (LpCopA), outlined a copper transport pathway across the membrane, which was inferred to be occluded. Here we show by
Yan Meng et al.
Biochimica et biophysica acta, 1690(3), 208-219 (2004-10-30)
Hepatic abnormalities in Long-Evans Cinnamon (LEC) rats, an animal model of Wilson disease (WD), were restored by the expression of the human ATP7B cDNA under the control of CAG promoter. Expression of ATP7B transcript and protein in the liver of
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