57697
Advanced Protein Assay
1× concentrate
Sinônimo(s):
Protein Assay, advanced
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About This Item
Código UNSPSC:
12171500
NACRES:
NA.51
Preço e disponibilidade não estão disponíveis no momento.
Produtos recomendados
Nível de qualidade
temperatura de armazenamento
2-8°C
Descrição geral
The Advanced Protein Assay Reagent combines the best properties of low protein-to-protein variance and a wide protein concentration, a sensitive detection limit and a unique red-to-blue color change.
Palavra indicadora
Warning
Frases de perigo
Declarações de precaução
Classificações de perigo
Acute Tox. 4 Oral - Met. Corr. 1 - STOT SE 2
Órgãos-alvo
Eyes,Central nervous system
Código de classe de armazenamento
8A - Combustible corrosive hazardous materials
Classe de risco de água (WGK)
WGK 2
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
Equipamento de proteção individual
Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter
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Romain Long et al.
Biochimica et biophysica acta, 1837(1), 201-209 (2013-10-29)
Carbon monoxide is continuously produced in small quantities in tissues and is an important signaling mediator in mammalian cells. We previously demonstrated that CO delivered to isolated rat heart mitochondria using a water-soluble CO-releasing molecule (CORM-3) is able to uncouple
Mathieu Panel et al.
The Journal of pharmacology and experimental therapeutics, 376(3), 348-357 (2020-12-12)
Mitochondrial permeability transition pore (mPTP) opening is a key event in cell death during myocardial ischemia reperfusion. Inhibition of its modulator cyclophilin D (CypD) by cyclosporine A (CsA) reduces ischemia-reperfusion injury. The use of cyclosporine A in this indication is
Stéphanie Paradis et al.
Cardiovascular research, 98(3), 420-427 (2013-04-05)
The translocator protein (TSPO) is located on the outer mitochondrial membrane where it is responsible for the uptake of cholesterol into mitochondria of steroidogenic organs. TSPO is also present in the heart where its role remains uncertain. We recently showed
Mathieu Panel et al.
Scientific reports, 7(1), 4283-4283 (2017-06-29)
Opening of the mitochondrial permeability transition pore (mPTP) plays a major role in cell death during cardiac ischaemia-reperfusion. Adult isolated rodent cardiomyocytes are valuable cells to study the effect of drugs targeting mPTP. This study investigated whether the use of
Matthias Kohlhauer et al.
Shock (Augusta, Ga.), 56(5), 857-864 (2021-05-13)
Mitochondria is often considered as the common nexus of cardiac and cerebral dysfunction after cardiac arrest. Here, our goal was to determine whether the time course of cardiac and cerebral mitochondrial dysfunction is similar after shockable versus non-shockable cardiac arrest
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