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SAB5600204

Sigma-Aldrich

Anti-beta-Actin antibody, Rabbit monoclonal

recombinant, expressed in HEK 293 cells, clone RM112, purified immunoglobulin

Synonym(e):

ACTB, Actin cytoplastic 1, actin beta, beta-actin

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About This Item

UNSPSC-Code:
12352203
NACRES:
NA.41

Rekombinant

expressed in HEK 293 cells

Antikörperform

purified immunoglobulin

Antikörper-Produkttyp

primary antibodies

Klon

RM112, monoclonal
recombinant monoclonal

Form

buffered aqueous glycerol solution

Speziesreaktivität

all

Methode(n)

ChIP: 1:200
ELISA: suitable (assay dependent concentration)
flow cytometry: suitable (assay dependent concentration)
immunocytochemistry: 1:200
immunohistochemistry: 1:200
immunoprecipitation (IP): 1:200
western blot: 1:1000

Isotyp

IgG

NCBI-Hinterlegungsnummer

60

UniProt-Hinterlegungsnummer

P60709

Versandbedingung

wet ice

Lagertemp.

−20°C

Posttranslationale Modifikation Target

unmodified

Angaben zum Gen

human ... ACTB(60)

Verwandte Kategorien

Spezifität

This antibody reacts to beta-Actin (Actin, cytoplasmic 1). No cross reactivity with other isoforms of Actin.

Immunogen

A peptide corresponding to the N-terminus of beta-actin

Physikalische Form

Solution in phosphate buffered saline containing 50% Glycerol/PBS with 1% BSA and 0.09% sodium azide.

Haftungsausschluss

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Lagerklassenschlüssel

10 - Combustible liquids

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

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Journal of controlled release : official journal of the Controlled Release Society, 349, 796-811 (2022-08-02)
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Scientific reports, 12(1), 10513-10513 (2022-06-23)
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Arthritis & rheumatology (Hoboken, N.J.), 74(4), 654-664 (2021-11-09)
Salivary gland epithelial cells (SGECs) are key cellular drivers in the pathogenesis of primary Sjögren's syndrome (SS); however, the mechanisms sustaining SGEC activation in primary SS remain unclear. We undertook this study to determine the role of autophagy in the
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