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Merck

N6530

Sigma-Aldrich

N1 Medium Supplement (100×)

solution, sterile-filtered, suitable for cell culture

Synonym(e):

N1 Supplement

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About This Item

UNSPSC-Code:
12352207
NACRES:
NA.77

Sterilität

sterile-filtered

Form

solution

Methode(n)

cell culture | mammalian: suitable

Verunreinigungen

endotoxin, tested

Lagertemp.

2-8°C

Allgemeine Beschreibung

N1 Medium Supplement (100×) can be used as a supplement to produce complete N1 neural cell growth media or closely related media.

Anwendung

N1 Medium Supplement (100×) has been used as:
  • a component of the proliferation medium for culturing oligodendrocyte-like cells and neuroblastoma cells
  • a supplement in the Dulbecco′s modified Eagle medium (DMEM/F12) to culture human oligodendrocytes
  • a component of the α-minimum essential medium (MEM) for culturing induced primary retinal pigment epithelium (ipRPE) monolayers

Sonstige Hinweise

Contains 0.5mg/ml recombinant human insulin, 0.5mg/ml human transferrin (partially iron-saturated), 0.5μg/ml sodium selenite, 1.6mg/ml putrescine, and 0.73μg/ml progesterone.

Angaben zur Herstellung

Prepared in Earle′s Balanced Salt Solution (EBSS) without phenol red.

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Lagerklassenschlüssel

12 - Non Combustible Liquids

WGK

WGK 1

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)


Analysenzertifikate (COA)

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Die Dokumentenbibliothek aufrufen

Ludmila A Voloboueva et al.
Investigative ophthalmology & visual science, 46(11), 4302-4310 (2005-10-27)
To determine whether (R)-alpha-lipoic acid (LA) protects cultured human fetal retinal pigment epithelial (hfRPE) cells against oxidative injury and identify the pathways that may mediate protection. Cultured hfRPE cells were pretreated with various concentrations of LA for 14 to 16
Miguel Flores-Bellver et al.
Journal of extracellular vesicles, 10(13), e12165-e12165 (2021-11-10)
Age-related macular degeneration (AMD) is a leading cause of blindness worldwide. Drusen are key contributors to the etiology of AMD and the ability to modulate drusen biogenesis could lead to therapeutic strategies to slow or halt AMD progression. The mechanisms
Lucia Lichvarova et al.
Scientific reports, 10(1), 12518-12518 (2020-07-23)
An amendment to this paper has been published and can be accessed via a link at the top of the paper.
Michael Craigie et al.
Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology, 13(2), 126-142 (2017-11-22)
JC virus (JCV) is a human polyomavirus and the etiologic agent of the demyelinating disease progressive multifocal leukoencephalopathy (PML). PML is observed in patients with underlying immunocompromising conditions, suggesting that neuro-immune interactions between peripheral immune cells and neuro-glia play an
Nicholas W Kieran et al.
Journal of neuroinflammation, 19(1), 10-10 (2022-01-08)
Astrocytes are the most numerous glial cell type with important roles in maintaining homeostasis and responding to diseases in the brain. Astrocyte function is subject to modulation by microRNAs (miRs), which are short nucleotide strands that regulate protein expression in

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