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AB16311

Sigma-Aldrich

Anti-Nitric Oxide Synthase II Antibody

Chemicon®, from rabbit

Synonym(e):

NOS II, iNOS

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About This Item

UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

Biologische Quelle

rabbit

Qualitätsniveau

100

Antikörperform

affinity purified immunoglobulin

Antikörper-Produkttyp

primary antibodies

Klon

polyclonal

Aufgereinigt durch

affinity chromatography

Speziesreaktivität

mouse

Hersteller/Markenname

Chemicon®

Methode(n)

immunocytochemistry: suitable
western blot: suitable

NCBI-Hinterlegungsnummer

NM_000625.3
NM_153292.1

UniProt-Hinterlegungsnummer

P35228

Versandbedingung

wet ice

Posttranslationale Modifikation Target

unmodified

Spezifität

Reactive with mouse NOS-II (iNOS).

Immunogen

Synthetic peptide corresponding to amino acids 1131-1144 of mouse macrophage NOS, coupled to KLH.

Anwendung

This Anti-Nitric Oxide Synthase II Antibody is validated for use in WB, IC for the detection of Nitric Oxide Synthase II.
Western Blot: 5 μg/mL using chemiluminescent detection. Higher concentrations of the antibody may be required if colorimetric detection is used. Antibody dilution will also vary with the concentration of the antigen. Immunocytochemistry: Use at 2-5 μg/mL

Optimal working dilutions must be determined by end user.

Verlinkung

Replaces: AB1631

Sonstige Hinweise

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Rechtliche Hinweise

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Lagerklassenschlüssel

12 - Non Combustible Liquids

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Bleranda Zeka et al.
Acta neuropathologica communications, 4(1), 82-82 (2016-08-10)
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Beatriz Linillos-Pradillo et al.
International journal of molecular sciences, 24(18) (2023-09-28)
The liver is the organ responsible for the metabolism and detoxification of BPF, the BPA analogue that is replacing it in plastic-based products. It is not known whether BPF can trigger inflammatory responses via the NLRP3 inflammasome, which plays a
Jordon Dunham et al.
Journal of neuropathology and experimental neurology, 76(6), 467-478 (2017-05-16)
Oxidative damage and iron redistribution are associated with the pathogenesis and progression of multiple sclerosis (MS), but these aspects are not entirely replicated in rodent experimental autoimmune encephalomyelitis (EAE) models. Here, we report that oxidative burst and injury as well
Isabella Wimmer et al.
Acta neuropathologica communications, 7(1), 14-14 (2019-02-02)
Human inflammatory or neurodegenerative diseases, such as progressive multiple sclerosis (MS), occur on a background of age-related microglia activation and iron accumulation as well as pre-existing neurodegeneration. Most experimental models for CNS diseases, however, are induced in rodents, which are

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