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S2320

Sigma-Aldrich

Anti-SFRS2 antibody produced in rabbit

affinity isolated antibody, PBS solution, contains 0.09% sodium azide as preservative

Synonym(e):

Anti-Splicing Factor Arginine/serine-rich 2

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About This Item

MDL-Nummer:
MFCD07370501
UNSPSC-Code:
12352203

Biologische Quelle

rabbit

Konjugat

unconjugated

Antikörperform

affinity isolated antibody

Antikörper-Produkttyp

primary antibodies

Klon

polyclonal

Form

PBS solution

Mol-Gew.

antigen 25.6 kDa

Enthält

0.09% sodium azide as preservative

Speziesreaktivität

human

Methode(n)

immunohistochemistry: suitable
western blot: suitable

Lagertemp.

−20°C

Angaben zum Gen

human ... SFRS2(6427)
mouse ... Sfrs2(20382)
rat ... Sfrs2(494445)

Immunogen

synthetic peptide from the N-terminal region of SFRS2 conjugated to KLH.

Anwendung

Applications in which this antibody has been used successfully, and the associated peer-reviewed papers, are given below.
Western Blotting (1 paper)

Zielbeschreibung

SFRS2 (Splicing Factor Arginine/Serine-rich 2), alsoknown as PR264, SC35 (splicing component 35), and SRp30b, is a member of the SR protein family of splicing factors. SFRS2 regulates alternative splicing of pre-mRNA.

Haftungsausschluss

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Lagerklassenschlüssel

10 - Combustible liquids

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

Eyeshields, Gloves

Analysenzertifikate (COA)

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Die Dokumentenbibliothek aufrufen

Matthew J Yousefzadeh et al.
EBioMedicine, 36, 18-28 (2018-10-04)
Senescence is a tumor suppressor mechanism activated in stressed cells to prevent replication of damaged DNA. Senescent cells have been demonstrated to play a causal role in driving aging and age-related diseases using genetic and pharmacologic approaches. We previously demonstrated
Rayna Rosati et al.
Clinical cancer research : an official journal of the American Association for Cancer Research, 24(24), 6509-6522 (2018-09-07)
Testosterone suppression in prostate cancer is limited by serious side effects and resistance via restoration of androgen receptor (AR) functionality. ELK1 is required for AR-dependent growth in various hormone-dependent and castration-resistant prostate cancer models. The amino-terminal domain of AR docks
Jana Kralovicova et al.
Nucleic acids research, 42(12), 8161-8173 (2014-06-20)
Splice-switching oligonucleotides (SSOs) have been widely used to inhibit exon usage but antisense strategies that promote removal of entire introns to increase splicing-mediated gene expression have not been developed. Here we show reduction of INS intron 1 retention by SSOs

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