推薦產品
產品線
MISSION®
儲存溫度
−20°C
一般說明
The MISSION LightSwitch Luciferase Assay Reagent enables one-step reagent addition to measure luciferase reporter signal. The MISSION LightSwitch Luciferase Assay Reagent and the MISSION 3′UTR Lenti GoClone report constructs are a fully optimized reporter system that includes an improved reporter gene (RenSP), an optimized assay reagent, and a genome-wide collection of 3′UTR Lenti GoClone reporter constructs. The MISSION 3′UTR Lenti GoClone reporter constructs have been optimized for use with the MISSION LightSwitch Assay Reagent.
應用
法律資訊
LightSwitch is a trademark of Active Motif
MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany
SwitchGear Genomics is a trademark of SwitchGear Genomics
訊號詞
Danger
危險分類
Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Flam. Liq. 2 - STOT SE 1
儲存類別代碼
3 - Flammable liquids
閃點（°F）
51.8 °F
閃點（°C）
11 °C
Janosch Hildebrand et al.
The Journal of investigative dermatology, 131(1), 20-29 (2010-09-10)
Here, we report a comprehensive investigation of changes in microRNA (miRNA) expression profiles on human keratinocyte (HK) differentiation in vitro and in vivo. We have monitored expression patterns of 377 miRNAs during calcium-induced differentiation of primary HKs, and have compared
Federico Innocenti et al.
PLoS genetics, 7(5), e1002078-e1002078 (2011-06-04)
The discovery of expression quantitative trait loci ("eQTLs") can help to unravel genetic contributions to complex traits. We identified genetic determinants of human liver gene expression variation using two independent collections of primary tissue profiled with Agilent (n = 206) and Illumina
An NTD-associated polymorphism in the 3' UTR of MTHFD1L can affect disease risk by altering miRNA binding.
Minguzzi S
Human Mutation, 35(1), 96-104 (2014)
The PLA2R1-JAK2 pathway upregulates ERRa and its mitochondrial program to exert tumor-suppressive action.
Griveau A
Oncogene, 35(38), 5033-5042 (2016)
Janine M van Gils et al.
Nature immunology, 13(2), 136-143 (2012-01-11)
Atherosclerotic plaque formation is fueled by the persistence of lipid-laden macrophages in the artery wall. The mechanisms by which these cells become trapped, thereby establishing chronic inflammation, remain unknown. Here we found that netrin-1, a neuroimmune guidance cue, was secreted
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