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Merck

C7499

Sigma-Aldrich

CK-548

≥98% (HPLC)

别名:

2-(3-Bromophenyl)-3-(5-chloro-2-hydroxyphenyl)-4-thiazolidinone, CK-0993548

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About This Item

经验公式(希尔记法):
C15H11BrClNO2S
分子量:
384.68
MDL號碼:
分類程式碼代碼:
12352200
PubChem物質ID:
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化驗

≥98% (HPLC)

形狀

solid

儲存條件

desiccated

溶解度

DMSO: 24 mg/mL

儲存溫度

2-8°C

SMILES 字串

Oc1ccc(Cl)cc1N2C(SCC2=O)c3cccc(Br)c3

InChI

1S/C15H11BrClNO2S/c16-10-3-1-2-9(6-10)15-18(14(20)8-21-15)12-7-11(17)4-5-13(12)19/h1-7,15,19H,8H2

InChI 密鑰

KEGQNJITMFBVAC-UHFFFAOYSA-N

生化/生理作用

CK-548 inhibits the activity of actin-related protein (Arp)2/3 complex by inserting into the hydrophobic core of Arp3 and altering its conformation: inhibits

象形圖

Exclamation markEnvironment

訊號詞

Warning

危險聲明

防範說明

危險分類

Acute Tox. 4 Oral - Aquatic Acute 1

儲存類別代碼

13 - Non Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Renu Khasa et al.
Microbial pathogenesis, 149, 104565-104565 (2020-10-16)
Multiple membrane trafficking networks operate in the eukaryotic cell and are hijacked by viruses to establish infection. Recent studied have highlighted that viruses can exploit distinct pathways depending on the cell type. Japanese encephalitis virus (JEV), a neurotropic flavivirus, can
Renu Khasa et al.
The Journal of general virology, 100(2), 176-186 (2018-11-30)
Japanese encephalitis virus (JEV), a mosquito-borne flavivirus, is one of the leading global causes of virus-induced encephalitis. The infectious life-cycle of viruses is heavily dependent on the host membrane trafficking network. Here, we have performed a RNA-interference-based screen using a
Arnat Balabiyev et al.
Molecular biology of the cell, 31(18), 2002-2020 (2020-06-25)
Macrophage fusion resulting in the formation of multinucleated giant cells (MGCs) is a multistage process that requires many adhesion-dependent steps and involves the rearrangement of the actin cytoskeleton. The diversity of actin-based structures and their role in macrophage fusion is
Monika Hartmann et al.
Molecular cancer research : MCR, 13(5), 879-890 (2015-02-06)
Ectodomain cleavage (shedding) of transmembrane proteins by metalloproteases (MMP) generates numerous essential signaling molecules, but its regulation is not totally understood. CD44, a cleaved transmembrane glycoprotein, exerts both antiproliferative or tumor-promoting functions, but whether proteolysis is required for this is

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