Increasing evidence verified that oxidative stress and neuroinflammatory response exacerbates motor deficits and increases neuronal loss in several rodent models of Parkinson's disease. In the present study, we explore the neuroprotective effects of monascin in a rotenone-induced Parkinson's disease model
Monascin is a major yellow compound from red mold dioscorea. We investigated monascin to test whether this compound acts as an antidiabetic and antioxidative stress agent in diabetic rats and Caenorhabditis elegans. The mechanisms by which monascin exerts its action
We examined the effect of various carbohydrates on the proliferative response of murine thymocytes to cytokines. The monosaccharide, D-mannose (4-10 mM), significantly inhibited thymocyte proliferation induced by recombinant interleukin-1 (rIL-1). Mannose also inhibited the proliferation response to native, human IL-1.
Malignant melanoma.
World journal of surgery, 16(2), 155-286 (1992-03-01)
[Symposium "Mechanisms of allergic inflammation". 2d Meeting of the Professional Society of the ADF: Immunologic mechanisms of inflammatory skin diseases. 21-23 February 1992, Freiburg i. Br].
W Czech
Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete, 43(11), 743-744 (1992-11-01)
