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36733

Supelco

(−)-Nikotin

PESTANAL®, analytical standard

Synonym(e):

(−)-1-Methyl-2-(3-pyridyl)pyrrolidin, (S)-3-(1-Methyl-2-pyrrolidinyl)pyridin, L-(−)-3-(N-Methyl)-α-pyrrolidyl)-pyridin

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About This Item

Empirische Formel (Hill-System):
C10H14N2
CAS-Nummer:
54-11-5
Molekulargewicht:
162.23
Beilstein:
82109
EG-Nummer:
200-193-3
MDL-Nummer:
MFCD00006369
UNSPSC-Code:
41116107
PubChem Substanz-ID:
329755481
NACRES:
NA.24

Qualität

analytical standard

Qualitätsniveau

100

Produktlinie

PESTANAL®

Haltbarkeit

limited shelf life, expiry date on the label

Methode(n)

HPLC: suitable
gas chromatography (GC): suitable

Brechungsindex

n20/D 1.5265 (lit.)

Dichte

1.010 g/mL at 20 °C (lit.)

Anwendung(en)

agriculture
cleaning products
cosmetics
environmental
food and beverages
personal care

Format

neat

SMILES String

CN1CCC[C@H]1c2cccnc2

InChI

1S/C10H14N2/c1-12-7-3-5-10(12)9-4-2-6-11-8-9/h2,4,6,8,10H,3,5,7H2,1H3/t10-/m0/s1

InChIKey

SNICXCGAKADSCV-JTQLQIEISA-N

Angaben zum Gen

human ... CHRNA2(1135) , CHRNA4(1137) , CHRNB2(1141)

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Verwandte Kategorien

Anwendung

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

Biochem./physiol. Wirkung

Prototyp Nikotin-Acetylcholin-Rezeptor-Agonist; natürlich vorkommendes Isomer.

Rechtliche Hinweise

PESTANAL is a registered trademark of Merck KGaA, Darmstadt, Germany

Signalwort

Danger

Gefahreneinstufungen

Acute Tox. 1 Oral - Acute Tox. 2 Dermal - Acute Tox. 2 Inhalation - Aquatic Chronic 2 - Eye Dam. 1 - Skin Irrit. 2

Lagerklassenschlüssel

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

WGK

WGK 3

Flammpunkt (°F)

213.8 °F - closed cup

Flammpunkt (°C)

101 °C - closed cup

Persönliche Schutzausrüstung

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter

Choose from one of the most recent versions:

Analysenzertifikate (COA)

Lot/Batch Number
BCCH6746
BCBW4394
BCBS3296V
BCBS3296
SZBE205XV

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USP

USP

1463382

Nikotin-verwandte Verbindung G

Nicotine solution NMR reference standard, 20 mg/mL in acetone-d6 (99.9 atom % D), TMS 0.05 %, CFCl3 0.4 %, NMR tube size 5 mm × 8 in.

Supelco

613223

Nicotine solution

Ramkrishna Mehendale et al.
American journal of obstetrics and gynecology, 197(4), 363-363 (2007-10-02)
Excess soluble vascular endothelial growth factor receptor, fms-like tyrosine kinase-1 (sFlt-1), and reduced placental growth factor (PlGF) mediate the genesis of preeclampsia. Cigarette smoking reduces the risk of preeclampsia. We hypothesized that placental secretion of sFlt-1 and PlGF was affected
Kang Li et al.
Annals of translational medicine, 9(9), 805-805 (2021-07-17)
Atherosclerosis is a chronic disease, with smoking being an independent risk factor. Irisin, a factor produced by myocytes, is expected to treat smoking-related arteriosclerosis, however its specific mechanism remains unclear. Forty Apoe-/- mice with nicotine intervention were involved in this
Pooja Naik et al.
BMC neuroscience, 15, 51-51 (2014-04-24)
Both active and passive tobacco smoke (TS) potentially impair the vascular endothelial function in a causative and dose-dependent manner, largely related to the content of reactive oxygen species (ROS), nicotine, and pro-inflammatory activity. Together these factors can compromise the restrictive
Charles C Horn et al.
American journal of physiology. Gastrointestinal and liver physiology, 298(1), G25-G30 (2009-11-07)
Nausea and emesis are common side effects of gastrointestinal disease. Reports indicate that ghrelin and endocannabinoids, agents that stimulate appetite, also reduce emesis evoked by chemotherapy treatment, which suggests that stimulation of feeding inhibits the emetic system. In the following
Charles C Horn et al.
American journal of physiology. Regulatory, integrative and comparative physiology, 306(5), R341-R351 (2014-01-17)
Signals from the vestibular system, area postrema, and forebrain elicit nausea and vomiting, but gastrointestinal (GI) vagal afferent input arguably plays the most prominent role in defense against food poisoning. It is difficult to determine the contribution of GI vagal
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