E4142
ExtrAvidin®−Cy3™
buffered aqueous solution
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1 ML
R$ 2.527,00
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1 ML
R$ 2.527,00
About This Item
Produtos recomendados
conjugado
CY3 conjugate
Formulário
buffered aqueous solution
Extensão da rotulagem
3-9 mol Cy3 per mol protein
técnica(s)
immunohistochemistry (formalin-fixed, paraffin-embedded sections): 1:100
Condições de expedição
wet ice
temperatura de armazenamento
2-8°C
Aplicação
Ações bioquímicas/fisiológicas
Avidin is a tetrameric or dimeric biotin-binding protein produced in the oviducts of birds, reptiles and amphibians and deposited in the whites of their eggs.[3] It consists of four high affinity binding sites for biotin. ExtrAvidin® is prepared from egg white avidin. It is a modified form of affinity purified avidin and binds biotin with high specific activity of avidin with the low background staining of streptavidin, a biotin binding protein produced by the bacteria Streptomyces avidinii.[4] ExtrAvidin® is covalently conjugated to Cy3 dye and is used for various techniques.
forma física
Solution in 0.01 M phosphate buffered saline, containing 1% bovine serum albumin and 15 mM sodium azide
Nota de preparo
Affinity purified protein
Informações legais
Cy3 is a trademark of Cytiva
ExtrAvidin is a registered trademark of Merck KGaA, Darmstadt, Germany
Exoneração de responsabilidade
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Código de classe de armazenamento
12 - Non Combustible Liquids
Classe de risco de água (WGK)
WGK 3
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
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A Signore et al.
Diabetologia, 40(12), 1476-1479 (1998-02-03)
The non-obese diabetic (NOD) mouse is widely used to study the pathogenesis of insulin-dependent diabetes mellitus. However, the mechanisms responsible for beta-cell destruction, in this model, are still poorly defined. The CD95/CD95L system among other effector systems has been implicated
L A Ballsmider et al.
Neural plasticity, 2015, 601985-601985 (2015-02-28)
This study investigated the anatomical integrity of vagal innervation of the gastrointestinal tract following vertical sleeve gastrectomy (VSG) and Roux-en-Y gastric bypass (RYGB) operations. The retrograde tracer fast blue (FB) was injected into the stomach to label vagal neurons originating
Andrea-Romana Prusa et al.
Human reproduction (Oxford, England), 18(7), 1489-1493 (2003-07-02)
It is the hope of investigators and patients alike that in future the isolation of pluripotent human stem cells will allow the establishment of therapeutic concepts for a wide variety of diseases. A major aim in this respect is the
Roux‑en‑Y gastric bypass surgery triggers rapid DNA fragmentation in vagal afferent neurons in rats.
Dulce M Minaya et al.
Acta neurobiologiae experimentalis, 79(4), 432-444 (2019-12-31)
Previous studies have shown that Roux‑en‑Y gastric bypass (RYGB), one of the most effective weight loss treatments for obesity, results in neurodegenerative responses in vagal afferent gut‑brain connection reflected by microglia activation and reduced sensory input to the nucleus tractus
Nisha V Shah et al.
Journal of ophthalmology, 2014, 829879-829879 (2014-04-16)
Purpose. To measure the chemotherapeutic effects of focal melphalan (intravitreal and subconjunctival) on tumor burden, hypoxia, and vasculature in LHBETATAG murine retinoblastoma model. Methods. LHBETATAG transgenic mice were treated with a single 1 mcg intravitreal injection of melphalan, 100 mcg subconjunctival
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