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15862
Cobalt chloride 0.1 M solution
Sinônimo(s):
Cobalt(II)-chloride solution, Additive Screening Solution 24/Kit-No 78374
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About This Item
Número CAS:
Número MDL:
Código UNSPSC:
12352302
ID de substância PubChem:
NACRES:
NA.32
Preço e disponibilidade não estão disponíveis no momento.
Produtos recomendados
grau
for analytical purposes
densidade
1.01 g/mL at 20 °C
temperatura de armazenamento
2-8°C
cadeia de caracteres SMILES
Cl[Co]Cl
InChI
1S/2ClH.Co/h2*1H;/q;;+2/p-2
chave InChI
GVPFVAHMJGGAJG-UHFFFAOYSA-L
Aplicação
Ações bioquímicas/fisiológicas
Cobalt chloride induces hypoxia condition in cells by upregulating hypoxia-inducible factor-1α (HIF-1α), erythropoietin and glycolytic enzymes. It is also responsible for mitochondrial DNA damage in rat neuronal PC12 cells.[3] It is also responsible for the induction of apoptosis.[4]
Cobalt chloride 0.1M solution is an additive screening solution of Additive Screening Kit. Additive Screen kit is designed to allow rapid and convenient evaluation of additives and their ability to influence the crystallization of the sample. The Additive Kit provides a tool for refining crystallization conditions.
Cobalt chloride 0.1M solution is an additive screening solution of Additive Screening Kit. Additive Screen kit is designed to allow rapid and convenient evaluation of additives and their ability to influence the crystallization of the sample. The Additive Kit provides a tool for refining crystallization conditions.
Palavra indicadora
Danger
Frases de perigo
Declarações de precaução
Classificações de perigo
Aquatic Chronic 2 - Carc. 1B - Repr. 1B
Código de classe de armazenamento
6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects
Classe de risco de água (WGK)
WGK 3
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
Equipamento de proteção individual
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
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Downregulation of duodenal SLC transporters and activation of proinflammatory signaling constitute the early response to high altitude in humans.
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Retinal hypoxia is one of the causative factors of diabetic retinopathy and is also one of the triggers of VEGF release. We hypothesized that specific dysregulated miRNAs in diabetic retinopathy could be linked to hypoxia-induced damage in human retinal endothelial
Mitochondrial DNA damage and a hypoxic response are induced by CoCl(2) in rat neuronal PC12 cells.
Wang G, et al.
Nucleic Acids Research, 28, 2135-2135 (2000)
CoCl(2) induces apoptosis via the 18 kDa translocator protein in U118MG human glioblastoma cells.
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Exposure to hypoxia following irradiation increases radioresistance in prostate cancer cells.
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