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MAB5554

Sigma-Aldrich

Anti-Pax6 Antibody, clone AD1.5

ascites fluid, clone AD1.5, Chemicon®

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About This Item

Código UNSPSC:
12352203
eCl@ss:
32160702
NACRES:
NA.41

fonte biológica

mouse

Nível de qualidade

100

forma do anticorpo

ascites fluid

tipo de produto de anticorpo

primary antibodies

clone

AD1.5, monoclonal

reatividade de espécies

zebrafish, rat, human, chicken, mouse

fabricante/nome comercial

Chemicon®

técnica(s)

immunohistochemistry: suitable
western blot: suitable

Isotipo

IgG1

nº de adesão NCBI

NM_000280.2
NM_001604.3

nº de adesão UniProt

P26367

Condições de expedição

dry ice

modificação pós-traducional do alvo

unmodified

Informações sobre genes

human ... PAX6(5080)

Especificidade

Recognizes Pax6.

Imunogênio

Recombinant human Pax6.

Aplicação

Anti-Pax6 Antibody, clone AD1.5 is an antibody against Pax6 for use in IH & WB.
Research Category
Neuroscience
Research Sub Category
Developmental Neuroscience

Neuronal & Glial Markers
Western blot. The antibody recognizes the 46 and 48 kDa Pax6 proteins.Immunohistochemistry on frozen tissue sections.Optimal working dilutions must be determined by end user.

Descrição-alvo

46 & 48 kDa

forma física

Ascites fluid containing no preservatives.
Unpurified

Armazenamento e estabilidade

Maintain for 1 year at -20°C from date of shipment. Aliquot to avoid repeated freezing and thawing. For maximum recovery of product, centrifuge the original vial after thawing and prior to removing the cap.

Nota de análise

Control
Embryonic eye tissue

Outras notas

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
This product can not be purchased for resale.

Informações legais

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Exoneração de responsabilidade

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Código de classe de armazenamento

10 - Combustible liquids

Classe de risco de água (WGK)

WGK 1

Ponto de fulgor (°F)

Not applicable

Ponto de fulgor (°C)

Not applicable

Certificados de análise (COA)

Busque Certificados de análise (COA) digitando o Número do Lote do produto. Os números de lote e remessa podem ser encontrados no rótulo de um produto após a palavra “Lot” ou “Batch”.

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Sergey Malchenko et al.
Gene, 534(2), 400-407 (2013-08-21)
In vitro neural differentiation of human embryonic stem cells (hESCs) is an advantageous system for studying early neural development. The process of early neural differentiation in hESCs begins by initiation of primitive neuroectoderm, which is manifested by rosette formation, with
Caroline A Johnson et al.
Development (Cambridge, England), 147(4) (2020-02-01)
Cellular and molecular mechanisms underlying the switch from self-amplification of cortical stem cells to neuronal and glial generation are incompletely understood, despite their importance for neural development. Here, we have investigated the role of the transcription factor specificity protein 2
Louise Thiry et al.
Communications biology, 7(1), 238-238 (2024-02-29)
The fatal motor neuron (MN) disease Amyotrophic Lateral Sclerosis (ALS) is characterized by progressive MN degeneration. Phrenic MNs (phMNs) controlling the activity of the diaphragm are prone to degeneration in ALS, leading to death by respiratory failure. Understanding of the
Christen M Crosta et al.
Molecular and cellular neurosciences, 109, 103562-103562 (2020-09-29)
Abnormal dendritic arbor development has been implicated in a number of neurodevelopmental disorders, such as autism and Rett syndrome, and the neuropsychiatric disorder schizophrenia. Postmortem brain samples from subjects with schizophrenia show elevated levels of NOS1AP in the dorsolateral prefrontal
Ramsey Najm et al.
Cell reports, 32(4), 107962-107962 (2020-07-30)
Despite its clear impact on Alzheimer's disease (AD) risk, apolipoprotein (apo) E4's contributions to AD etiology remain poorly understood. Progress in answering this and other questions in AD research has been limited by an inability to model human-specific phenotypes in
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