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MAB1548

Sigma-Aldrich

Anti-Myosin Antibody, heavy chain β

culture supernatant, clone 5B9 (2C8), Chemicon®

Sinônimo(s):

Anti-CMD1S, Anti-MPD1, Anti-MYHCB, Anti-SPMD, Anti-SPMM

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About This Item

Código UNSPSC:
12352203
eCl@ss:
32160702
NACRES:
NA.41

fonte biológica

mouse

Nível de qualidade

forma do anticorpo

culture supernatant

tipo de produto de anticorpo

primary antibodies

clone

5B9 (2C8), monoclonal

reatividade de espécies

human

fabricante/nome comercial

Chemicon®

técnica(s)

immunohistochemistry: suitable
western blot: suitable

Isotipo

IgG2a

nº de adesão NCBI

nº de adesão UniProt

Condições de expedição

wet ice

modificação pós-traducional do alvo

unmodified

Informações sobre genes

rat ... Myh7(29557)

Especificidade

Recognizes the S1/S2 junction of human ventricular myosin heavy chain beta

Imunogênio

Epitope: heavy chain beta
Human ventricular myosin

Aplicação

Detect Myosin using this Anti-Myosin Antibody, heavy chain β validated for use in WB, IH.
Immunohistochemistry: Neat (undiluted)
Western blot: 1:10

Optimal dilutions must be determined by the end user.

forma física

Supernatant in RPMI 1640, 10% FCS, 0.01% sodium azide.

Armazenamento e estabilidade

Maintain at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.

Informações legais

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Código de classe de armazenamento

12 - Non Combustible Liquids

Classe de risco de água (WGK)

WGK 2

Ponto de fulgor (°F)

Not applicable

Ponto de fulgor (°C)

Not applicable


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Barbara S Mallon et al.
Stem cell research, 12(2), 376-386 (2014-01-01)
Many studies have compared the genetic and epigenetic profiles of human induced pluripotent stem cells (hiPSCs) to human embryonic stem cells (hESCs) and yet the picture remains unclear. To address this, we derived a population of neural precursor cells (NPCs)
Siva K Panguluri et al.
American journal of physiology. Heart and circulatory physiology, 304(12), H1651-H1661 (2013-04-16)
Ventricular arrhythmias account for high mortality in cardiopulmonary patients in intensive care units. Cardiovascular alterations and molecular-level changes in response to the commonly used oxygen treatment remains unknown. In the present study we investigated cardiac hypertrophy and cardiac complications in
Henry E Young et al.
The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology, 276(1), 75-102 (2003-12-31)
Development of a multicellular organism is accomplished through a series of events that are preprogrammed in the genome. These events encompass cellular proliferation, lineage commitment, lineage progression, lineage expression, cellular inhibition, and regulated apoptosis. The sequential progression of cells through
Henry E Young et al.
The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology, 277(1), 178-203 (2004-02-26)
Undifferentiated cells have been identified in the prenatal blastocyst, inner cell mass, and gonadal ridges of rodents and primates, including humans. After isolation these cells express molecular and immunological markers for embryonic cells, capabilities for extended self-renewal, and telomerase activity.
Richard P Davis et al.
Circulation, 125(25), 3079-3091 (2012-06-01)
Pluripotent stem cells (PSCs) offer a new paradigm for modeling genetic cardiac diseases, but it is unclear whether mouse and human PSCs can truly model both gain- and loss-of-function genetic disorders affecting the Na(+) current (I(Na)) because of the immaturity

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