341272
Anti-Factor B Goat pAb
liquid, Calbiochem®
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About This Item
Código UNSPSC:
12352203
NACRES:
NA.43
Preço e disponibilidade não estão disponíveis no momento.
Produtos recomendados
fonte biológica
goat
Nível de qualidade
forma do anticorpo
serum
tipo de produto de anticorpo
primary antibodies
clone
polyclonal
Formulário
liquid
contém
≤0.1% sodium azide as preservative
reatividade de espécies
human
fabricante/nome comercial
Calbiochem®
disponibilidade
not available in Canada
condição de armazenamento
OK to freeze
avoid repeated freeze/thaw cycles
Isotipo
IgG
Condições de expedição
ambient
temperatura de armazenamento
−20°C
modificação pós-traducional do alvo
unmodified
Informações sobre genes
human ... CFB(629)
Descrição geral
Goat polyclonal antibody supplied as undiluted serum. Recognizes Factor B.
Recognizes Factor B.
This Anti-Factor B Goat pAb is validated for use in Immunoelectrophoresis, Radial Immunodiffusion, Ouchterlony for the detection of Factor B.
Imunogênio
Human
purified, human factor B
Aplicação
Immunoelectrophoresis (see comments)
Radial Immunodiffusion (see comments)
Ouchterlony (≥ undiluted)
Radial Immunodiffusion (see comments)
Ouchterlony (≥ undiluted)
Embalagem
Please refer to vial label for lot-specific concentration.
Advertência
Toxicity: Standard Handling (A)
Reconstituição
Following initial thaw, aliquot and freeze (-20°C).
Outras notas
This antibody has also been reported to work for immunoelectrophoresis and radial immunodiffusion. Variables associated with assay conditions will dictate the proper working dilution.
Informações legais
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
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Código de classe de armazenamento
10 - Combustible liquids
Classe de risco de água (WGK)
WGK 1
Certificados de análise (COA)
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Encontre a documentação dos produtos que você adquiriu recentemente na biblioteca de documentos.
Anna M Blom et al.
Journal of clinical immunology, 36(5), 517-527 (2016-05-06)
Autoantibodies termed C3-nephritic factor (C3NeF), which stabilize convertases of the alternative complement pathway, often stimulate autoinflammatory diseases. However, knowledge about analogous autoantibodies acting on the classical pathway (C4NeF) is limited to a few reports, which indicate association with kidney dysfunction
C Duehrkop et al.
Laboratory animals, 48(2), 143-154 (2014-03-04)
Prolonged ischemia of skeletal muscle tissue, followed by reperfusion, leads to ischemia/reperfusion injury (IRI), which is a feared local and systemic inflammatory reaction. With respect to the 3Rs, we wanted to determine which parameters for assessment of IRI require a
Claudia Duehrkop et al.
PloS one, 8(8), e72059-e72059 (2013-08-31)
Ischemia/reperfusion injury of lower extremities and associated lung damage may result from thrombotic occlusion, embolism, trauma, or surgical intervention with prolonged ischemia and subsequent restoration of blood flow. This clinical entity is characterized by high morbidity and mortality. Deprivation of
Diana Pauly et al.
Cell reports, 29(9), 2835-2848 (2019-11-28)
Complement dysregulation is a feature of many retinal diseases, yet mechanistic understanding at the cellular level is limited. Given this knowledge gap about which retinal cells express complement, we performed single-cell RNA sequencing on ∼92,000 mouse retinal cells and validated
Filippo Mori et al.
Frontiers in immunology, 15, 1334151-1334151 (2024-06-26)
Complement factor H (FH) is a major regulator of the complement alternative pathway, its mutations predispose to an uncontrolled activation in the kidney and on blood cells and to secondary C3 deficiency. Plasma exchange has been used to correct for
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