MAB333
Anti-Synaptobrevin Antibody, clone SP10
ascites fluid, clone SP10, Chemicon®
Synonym(e):
VAMP
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100 μL
€ 324,00
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100 μL
€ 324,00
About This Item
UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Empfohlene Produkte
Biologische Quelle
mouse
Qualitätsniveau
Antikörperform
ascites fluid
Antikörper-Produkttyp
primary antibodies
Klon
SP10, monoclonal
Speziesreaktivität
rat, hamster, salamander, pig, human
Hersteller/Markenname
Chemicon®
Methode(n)
immunohistochemistry: suitable
western blot: suitable
Isotyp
IgM
NCBI-Hinterlegungsnummer
UniProt-Hinterlegungsnummer
Versandbedingung
wet ice
Posttranslationale Modifikation Target
unmodified
Angaben zum Gen
human ... VAMP1(6843)
Spezifität
Synaptobrevin [VAMP]. MAB333 reacts with VAMP-1 and VAMP-2 fusion protein expressed in bacterial system.
Immunogen
Crude synaptic immunoprecipitate (Human).
Anwendung
Research Category
Neurowissenschaft
Neurowissenschaft
Research Sub Category
Synapsen & Synapsenbiologie
Synapsen & Synapsenbiologie
Anti-Synaptobrevin Antibody, clone SP10 is an antibody against Synaptobrevin for use in WB, IH.
Immunohistochemistry at >1:25.
Western blotting at 1:100-1:10,000.
Optimal working dilutions must be determined by the end user.
Western blotting at 1:100-1:10,000.
Optimal working dilutions must be determined by the end user.
Verlinkung
Replaces: AB5863P
Physikalische Form
Liquid, contains sodium azide.
Lagerung und Haltbarkeit
Maintain frozen at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.
Rechtliche Hinweise
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Haftungsausschluss
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Lagerklassenschlüssel
10 - Combustible liquids
Analysenzertifikate (COA)
Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.
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G Bonanno et al.
British journal of pharmacology, 129(8), 1780-1786 (2000-04-26)
The release of [(3)H]-dopamine ([(3)H]-DA) from human neocortex nerve terminals was studied in synaptosomes prepared from brain specimens removed in neurosurgery and exposed during superfusion to different releasing stimuli. Treatment with 15 mM KCl, 100 microM 4-aminopyridine, 1 microM ionomycin
Huayu Qi et al.
Molecular biology of the cell, 13(2), 530-541 (2002-02-21)
The zona pellucida (ZP) is a highly organized extracellular coat that surrounds all mammalian eggs. The mouse egg ZP is composed of three glycoproteins, called mZP1-3, that are synthesized, secreted, and assembled into a ZP exclusively by growing oocytes. Here
M Raiteri et al.
Journal of neurochemistry, 74(1), 423-431 (2000-01-05)
Small molecules present during brain tissue homogenization are known to be entrapped within subsequently isolated synaptosomes. We have revisited this technique in view of its systematic utilization to incorporate into nerve endings impermeant probes of large size. Rat neocortical synaptosomes
Elizabeth Scarr et al.
Clinical psychopharmacology and neuroscience : the official scientific journal of the Korean College of Neuropsychopharmacology, 10(1), 25-33 (2013-02-23)
Given the ability of mood stabilizers and antipsychotics to promote cell proliferation, we wanted to determine the effects of these drugs on neuronal markers previously reported to be altered in subjects with psychiatric disorders. Male Sprauge-Dawley rats were treated with
Richard Henriksson et al.
Synapse (New York, N.Y.), 62(11), 829-833 (2008-08-23)
Convergent lines of evidence suggest potentiation of glutamatergic synapses after chronic ethanol exposure, and indicate that the presynaptic effect hereof is on modulators of synaptic strength rather than on executors of glutamate release. To address this hypothesis in the context
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