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06720

Sigma-Aldrich

2-Aminoethyl hydrogen sulfate

≥98.0% (T)

Synonym(s):

Sulfuric acid mono 2-aminoethylester

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100 G
$80.90
500 G
$98.00

About This Item

Linear Formula:
NH2CH2CH2OSO3H
CAS Number:
Molecular Weight:
141.15
Beilstein/REAXYS Number:
1704079
EC Number:
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.22

$80.90


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assay

≥98.0% (T)

mp

277 °C (dec.) (lit.)

functional group

amine

SMILES string

NCCOS(O)(=O)=O
NCCOS(O)(=O)=O

InChI

1S/C2H7NO4S/c3-1-2-7-8(4,5)6/h1-3H2,(H,4,5,6)

InChI key

WSYUEVRAMDSJKL-UHFFFAOYSA-N

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pictograms

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signalword

Warning

Hazard Classifications

Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

dust mask type N95 (US), Eyeshields, Gloves


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T Táira et al.
Psychopharmacology, 109(1-2), 191-197 (1992-01-01)
Long-term effects of chronic treatment with a GABA-T (GABA-transaminase) inhibitor, ethanolamine O-sulphate (EOS) (200 mg/kg/day for the postnatal days 3-21) on the binding parameters of GABAA receptors, hypothalamic monoamines and subsequent behavior were studied in Wistar rats. At the age
P Baud et al.
Brain research, 452(1-2), 203-211 (1988-06-14)
Locomotor activity in the rat was studied after infusion of GABAergic and enkephalinergic agonists into the nucleus basalis magnocellularis (NBM) of the forebrain. The experiments were designed to find out whether pharmacological blockade of cholinergic neurons in the NBM had
M Qume et al.
British journal of pharmacology, 122(3), 539-545 (1997-11-14)
1. The effects of 2, 8 and 21 day oral treatment with the specific gamma-aminobutyric acid transaminase (GABA-T) inhibitors gamma-vinyl GABA (GVG) and ethanolamine O-sulphate (EOS) on brain GABA levels, GABA-T activity, and basal and stimulated GABA release from rat
A E Herbison et al.
Journal of neurochemistry, 55(5), 1617-1623 (1990-11-01)
The characteristics of gamma-aminobutyric acid (GABA) release as monitored by microdialysis have been investigated in the chloral hydrate anaesthetised rat. The high outflow of GABA following insertion of the microdialysis probe (membrane 2 mm in length, 0.5 mm in diameter)
M Qume et al.
Biochemical pharmacology, 52(9), 1355-1363 (1996-11-08)
The inhibitory neurotransmitter gamma-aminobutyric acid (GABA) is not solely located in the CNS, it and the enzymes responsible for its synthesis (glutamic acid decarboxylase, GAD, EC 4.1.1.15) and catabolism (GABA-transaminase, GABA-T, EC 2.6.1.19) are also present in non-neuronal organs. Following

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