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Merck
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重要文件

M0895

Sigma-Aldrich

MES 钾盐

≥99% (titration)

同義詞:

2-(N-吗啉基)乙磺酸 钾盐, 4-吗啉乙磺酸 钾盐

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About This Item

經驗公式(希爾表示法):
C6H12KNO4S
CAS號碼:
分子量::
233.33
MDL號碼:
分類程式碼代碼:
12161700
eCl@ss:
32129211
PubChem物質ID:
NACRES:
NA.25
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化驗

≥99% (titration)

形狀

crystalline powder

有用的pH值範圍

5.5-6.7

pKa 

6.1

溶解度

water: 0.33 g/mL, clear, colorless

SMILES 字串

[K+].[O-]S(=O)(=O)CCN1CCOCC1

InChI

1S/C6H13NO4S.K/c8-12(9,10)6-3-7-1-4-11-5-2-7;/h1-6H2,(H,8,9,10);/q;+1/p-1

InChI 密鑰

IMFIKFZWLAWMQE-UHFFFAOYSA-M

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應用

MES potassium salt has been used as a component of buffers for the preparation of permeabilized fiber bundles.[1][2]

生化/生理作用

MES is applicable as a Good′s buffer and is widely used in regulating the pH of reagent solutions, physiological experiments and plant culture medium.[3]

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

Eyeshields, Gloves, type N95 (US)


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Tomoko Kagenishi et al.
Frontiers in plant science, 7, 79-79 (2016-03-01)
In plants, growth of roots and root hairs is regulated by the fine cellular control of pH and reactive oxygen species (ROS). MES, 2-(N-morpholino)ethanesulfonic acid as one of the Good's buffers has broadly been used for buffering medium, and it
Cody D Smith et al.
The Journal of biological chemistry, 295(48), 16207-16216 (2020-08-05)
Compensatory changes in energy expenditure occur in response to positive and negative energy balance, but the underlying mechanism remains unclear. Under low energy demand, the mitochondrial electron transport system is particularly sensitive to added energy supply (i.e. reductive stress), which
Ashley S Williams et al.
Cell metabolism, 31(1), 131-147 (2019-12-10)
This study sought to examine the functional significance of mitochondrial protein acetylation using a double knockout (DKO) mouse model harboring muscle-specific deficits in acetyl-CoA buffering and lysine deacetylation, due to genetic ablation of carnitine acetyltransferase and Sirtuin 3, respectively. DKO
Trace Thome et al.
American journal of physiology. Cell physiology, 317(4), C701-C713 (2019-07-11)
Chronic kidney disease (CKD) leads to increased skeletal muscle fatigue, weakness, and atrophy. Previous work has implicated mitochondria within the skeletal muscle as a mediator of muscle dysfunction in CKD; however, the mechanisms underlying mitochondrial dysfunction in CKD are not

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