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Merck

32307

Sigma-Aldrich

醋酸铅 三水合物

puriss. p.a., ACS reagent, reag. ISO, reag. Ph. Eur., 99.5-102.0%

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About This Item

线性分子式:
Pb(CH3CO2)2 · 3H2O
CAS号:
分子量:
379.33
Beilstein:
3730298
EC號碼:
MDL號碼:
分類程式碼代碼:
12352302
PubChem物質ID:
NACRES:
NA.21

等級

ACS reagent
puriss. p.a.

品質等級

agency

USP/NF
reag. ISO
reag. Ph. Eur.

化驗

99.5-102.0%

形狀

solid

雜質

≤0.005% insoluble in acetic acid

mp

75 °C (dec.) (lit.)

負離子痕跡

chloride (Cl-): ≤5 mg/kg
nitrate (NO3-): ≤10 mg/kg

正離子痕跡

Ca: ≤50 mg/kg
Cd: ≤5 mg/kg
Cu: ≤5 mg/kg
Fe: ≤10 mg/kg
K: ≤50 mg/kg
Mg: ≤50 mg/kg
Na: ≤50 mg/kg
Zn: ≤5 mg/kg

SMILES 字串

O.O.O.CC(=O)O[PbH2]OC(C)=O

InChI

1S/2C2H4O2.3H2O.Pb/c2*1-2(3)4;;;;/h2*1H3,(H,3,4);3*1H2;/q;;;;;+2/p-2

InChI 密鑰

MCEUZMYFCCOOQO-UHFFFAOYSA-L

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一般說明

醋酸铅(II)是一种白色结晶化合物。它广泛应用于化妆品。醋酸铅试纸用于检测有毒气体、硫化氢。它可与5-乙炔基-1,3-苯二甲酸(H2ebdc)反应,生成一维梯形网络,[Pb(ebdc)(MeOH)]2·H2O。

應用

三水乙酸铅(II)可用于制备PbSe-CdSe-PbSe纳米哑铃 。

訊號詞

Danger

危險分類

Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 2 - Eye Dam. 1 - Lact. - Repr. 1A - STOT RE 1

標靶器官

Central nervous system,Blood,Immune system,Kidney

儲存類別代碼

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Hazardous Chemicals: Safety Management and Global Regulations, 244-244 (2013)
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Chemical communications (Cambridge, England), 50(92), 14436-14439 (2014-10-11)
5-Ethynyl-1,3-benzenedicarboxylic acid (H2ebdc) reacted with lead(II) acetate trihydrate yields a 1D ladder network, [Pb(ebdc)(MeOH)]2·H2O (1). Removing crystals of 1 from the mother liquor results in a facile single crystal to single crystal transition, yielding 2D [Pb(ebdc)] net (2) with a
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Clinical toxicology (Philadelphia, Pa.), 51(4), 225-229 (2013-03-27)
Occupational and environmental exposures to lead (Pb) are a worldwide concern. DNA methylation plays an important role in the development of Pb toxicity. Here, we try to find out the evidence to prove that the methylation of the LINE-1 promoter
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Toxicology letters, 220(1), 44-52 (2013-04-20)
The mechanisms by which early chronic lead (Pb) exposure alter brain development have not been identified. We examined neuroimmune system effects in C57BL/6J mice with Pb exposure, including levels that may be common among children in lower socioeconomic income environments.

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