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Merck
모든 사진(1)

주요 문서

690228

Sigma-Aldrich

N,N-Bis[3-(methylamino)propyl]methylamine

98%

동의어(들):

N-Methyl-3,3′-bis(methylamino)dipropylamine, N-Methyl-N,N-bis[3-(methylamino)propyl]amine, Methylbis(3-methylaminopropyl)amine

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About This Item

Linear Formula:
CH3N(CH2CH2CH2NHCH3)2
CAS Number:
Molecular Weight:
173.30
Beilstein:
6711228
EC Number:
MDL number:
UNSPSC 코드:
12352100
PubChem Substance ID:
NACRES:
NA.22

분석

≥97.5% (GC)
97.5-102.5% (with HClO4, T)
98%

양식

liquid

불순물

≤1% water

SMILES string

CNCCCN(C)CCCNC

InChI

1S/C9H23N3/c1-10-6-4-8-12(3)9-5-7-11-2/h10-11H,4-9H2,1-3H3

InChI key

PQYGBJHVVYREGU-UHFFFAOYSA-N

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픽토그램

Corrosion

신호어

Danger

유해 및 위험 성명서

Hazard Classifications

Skin Corr. 1B

Storage Class Code

8A - Combustible corrosive hazardous materials

WGK

WGK 3

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable

개인 보호 장비

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter


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문서 라이브러리 방문

D Nochlin et al.
Journal of neuropathology and experimental neurology, 41(2), 233-240 (1982-03-01)
The administration of an aliphatic triamine, 3,3'-methylimino-bis-(N-methylpropylamine), was used to produce non-necrotizing lesions in the hypothalamus and medulla oblongata of rats. The distribution of the lesions in the hypothalamus was similar to that produced by goldthioglucose, and consisted ultrastructurally of
Gold thioglucose, mimpa and lesions of the ventromedial hypothalamus.
D F Brown
Journal of neuropathology and experimental neurology, 43(4), 455-457 (1984-07-01)
S Levine et al.
Journal of immunological methods, 54(3), 355-360 (1982-11-12)
In the past, the lesions of experimental allergic encephalomyelitis (EAE) have been induced to localize around brain tissue damaged by anoxia or direct physical or chemical attack. The procedure for producing the requisite antecedent brain injury has been simplified by
S Levine et al.
Brain research, 242(2), 219-225 (1982-06-24)
An aliphatic triamine has been reported to cause lesions in rats in the vicinity of the area postrema and the median eminence of the hypothalamus, sites known to lack a blood-brain barrier. The present study revealed that some of the
D F Brown et al.
Physiology & behavior, 49(1), 41-46 (1991-01-01)
It has been demonstrated that the ventromedial hypothalamus (VMH) of alloxan-induced diabetic mice is protected from subsequent gold thioglucose (GTG)-induced lesions. Another compound, 3,3'-methyliminobis-(N-methylpropylamine) (MIMPA), a triamine structurally unrelated to GTG, has been shown to cause similar VMH lesions in

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