Wheat germ lectin, also known as wheat germ agglutinin (WGA), has a molecular weight of 36 kDa. It shows affinity to 1,4-N-acetylglucosamine (GlcNAc) oligomers and is useful in studying surface components of C. neoformans.
免疫原
コムギ胚芽レクチン(WGA)。
アプリケーション
Anti-Lectin, Triticum vulgaris antibody produced in rabbit has been used in
immunolabeling experiments
immunohistochemistry
immunocytochemistry
Anti-Lectin, Triticum vulgaris antibody produced in rabbit has been used in immunohistochemistry and immunocytochemistry.
生物化学的/生理学的作用
Wheat germ agglutinin is a lectin isolated from Triticum vulgaris that has affinity for chitin oligosaccharides, peptidoglycans and glycolipids via sialic acid residues. It is not specific for human blood groups and has been identified as antagonists of botulinum and tetanus toxins.
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The American journal of pathology, 187(2), 268-279 (2017-01-16)
Staphylococcus aureus causes very serious infections of vascular grafts. Knowledge of the molecular mechanisms of this disease is largely lacking because of the absence of representable models. Therefore, the aim of this study was to set up a mouse model
The Journal of comparative neurology, 519(13), 2648-2657 (2011-04-13)
We recently developed a genetic transneuronal tracing approach that allows for the study of circuits that are altered by nerve injury. We generated transgenic (ZW-X) mice in which expression of a transneuronal tracer, wheat germ agglutinin (WGA), is induced in
Spinal cord and peripheral nerve injury results in extensive damage to the locally injured cells as well as distant cells that are functionally connected to them. Both primary and secondary damage can cause a broad range of clinical abnormalities, including
Detection of local and remote cellular damage caused by spinal cord and peripheral nerve injury using a heat shock signaling reporter system
The Journal of comparative neurology, 507(6), 1990-2003 (2008-02-15)
Despite the evidence for a significant contribution of brainstem serotonergic (5HT) systems to the control of spinal cord "pain" transmission neurons, attention has turned recently to the influence of nonserotonergic neurons, including the facilitatory and inhibitory controls that originate from