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￥53,350
About This Item
実験式（ヒル表記法）:
Cu
CAS番号:
分子量:
63.55
MDL番号:
UNSPSCコード:
12141711
PubChem Substance ID:
NACRES:
NA.23
おすすめの製品
アッセイ
≥99.9%
フォーム
wire
メーカー／製品名
Goodfellow 977-785-58
抵抗性
1.673 μΩ-cm, 20°C
bp
2567 °C (lit.)
mp
1083.4 °C (lit.)
密度
8.94 g/mL at 25 °C (lit.)
SMILES記法
[Cu]
InChI
1S/Cu
InChI Key
RYGMFSIKBFXOCR-UHFFFAOYSA-N
詳細
For updated SDS information please visit www.goodfellow.com.
法的情報
Goodfellow製品
保管分類コード
13 - Non Combustible Solids
WGK
WGK 2
引火点(°F)
Not applicable
引火点(℃)
Not applicable
最新バージョンのいずれかを選択してください：
Donita C Brady et al.
Nature, 509(7501), 492-496 (2014-04-11)
The BRAF kinase is mutated, typically Val 600→Glu (V600E), to induce an active oncogenic state in a large fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide spectrum of other cancers. BRAF(V600E) phosphorylates and
Hiroki Serizawa et al.
Organic letters, 16(13), 3456-3459 (2014-06-14)
The direct synthesis of pentafluoroethyl copper (CuC2F5) from a cuprate reagent and ethyl pentafluoropropionate as one of the most economical and useful pentafluoroethyl sources was accomplished. The advantages of this method are; all the reagents employed are low-cost and operationally
Alina Fedoseienko et al.
Annals of the New York Academy of Sciences, 1314, 6-14 (2014-04-05)
Copper is an important cofactor in numerous biological processes in all living organisms. However, excessive copper can be extremely toxic, so it is vital that the copper level within a cell is tightly regulated. The damaging effect of copper is
Giulia Cheloni et al.
Aquatic toxicology (Amsterdam, Netherlands), 155, 275-282 (2014-07-30)
The present study showed the important role of light intensity and spectral composition on Cu uptake and effects on green alga Chlamydomonas reinhardtii. High-intenisty light (HL) increased cellular Cu concentrations, but mitigated the Cu-induced decrease in chlorophyll fluorescence, oxidative stress
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
アクティブなフィルタ
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