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Merck
Tutte le immagini(1)

Documenti fondamentali

S9384

Supelco

Suloctidil

analytical standard, for drug analysis

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About This Item

Formula empirica (notazione di Hill):
C20H35NOS
Numero CAS:
Peso molecolare:
337.56
Numero CE:
Numero MDL:
Codice UNSPSC:
41116107
ID PubChem:
NACRES:
NA.24

tecniche

HPLC: suitable
gas chromatography (GC): suitable

applicazioni

forensics and toxicology
pharmaceutical (small molecule)
veterinary

Formato

neat

Temperatura di conservazione

2-8°C

Stringa SMILE

CCCCCCCCN[C@H](C)[C@@H](O)c1ccc(SC(C)C)cc1

InChI

1S/C20H35NOS/c1-5-6-7-8-9-10-15-21-17(4)20(22)18-11-13-19(14-12-18)23-16(2)3/h11-14,16-17,20-22H,5-10,15H2,1-4H3/t17-,20-/m1/s1
BFCDFTHTSVTWOG-YLJYHZDGSA-N

Applicazioni

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

Pittogrammi

Exclamation mark

Avvertenze

Warning

Indicazioni di pericolo

Classi di pericolo

Acute Tox. 4 Oral

Codice della classe di stoccaggio

13 - Non Combustible Solids

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

dust mask type N95 (US), Eyeshields, Gloves


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Certificati d'analisi (COA)

Lot/Batch Number

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Combination of suloctidil and anticoagulation in the prevention of reocclusion after femoro-popliteal PTA.
F Mahler et al.
VASA. Zeitschrift fur Gefasskrankheiten, 16(4), 381-385 (1987-01-01)
P Kontro et al.
Neuroscience, 19(3), 1007-1010 (1986-11-01)
The effects of taurine and its structural analogues and two new anticonvulsant derivatives, taltrimide and MY-103, on the function of brain dopaminergic systems were studied by assessing their interference with the binding of [3H]spiperone to synaptic membranes isolated from rat
J M Boeynaems et al.
Biochemical pharmacology, 36(10), 1629-1635 (1987-05-15)
Suloctidil is a calcium antagonist with vascular relaxing activity and an antithrombotic agent: its antiplatelet action has been demonstrated in vivo, but is difficult to reproduce in vitro and the mechanism of this effect remains unknown. We have observed that
C Mazière et al.
Journal of clinical chemistry and clinical biochemistry. Zeitschrift fur klinische Chemie und klinische Biochemie, 26(1), 3-6 (1988-01-01)
Human foetal lung fibroblasts were pretreated for 24 h with the antithrombotic drug, suloctidil (1 to 10 mumol/l), which induced a dose-dependent increase in LDL binding, uptake and degradation. At 10 mumol/l suloctidil, the respective increases in these parameters were
P Calderon et al.
Archives internationales de pharmacodynamie et de therapie, 284(1), 101-113 (1986-11-01)
The vasorelaxing effect of suloctidil was evaluated in isolated rat and rabbit aorta and in isolated rabbit mesenteric and saphenous artery. Suloctidil inhibited contractions induced by increasing extracellular calcium in depolarized arteries, mainly in a competitive way. In the rat

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