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Merck

L3289

Sigma-Aldrich

血液裂解液

sufficient for 100 reactions, for molecular biology

别名:

Extract-N-Amp 直接PCR血裂解液

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About This Item

分類程式碼代碼:
12352200
NACRES:
NA.25

等級

for molecular biology

形狀

liquid

用途

sufficient for 100 reactions

儲存溫度

−20°C

一般說明

血液裂解溶液可结合血液中和溶液 (N9784)一起使用, 有助于从全血、血卡上干燥的全血和培养的哺乳动物细胞中快速提取和中和 DNA。

该裂解溶液是Extract-N-Amp和REDExtract-N-Amp血液PCR试剂盒的组分

應用

血液裂解液用于在聚合酶链反应 (PCR) 扩增以前裂解血液细胞以提取DNA。它还可用于分离 CRISPR-Cas9 基因编辑的基因组 DNA。

法律資訊

Extract-N-Amp is a trademark of Sigma-Aldrich Co. LLC
REDExtract-N-Amp is a trademark of Sigma-Aldrich Co. LLC

象形圖

Corrosion

訊號詞

Danger

危險聲明

危險分類

Eye Dam. 1 - Met. Corr. 1 - Skin Corr. 1B

儲存類別代碼

8A - Combustible corrosive hazardous materials

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Identification by random mutagenesis of functional domains in KREPB5 that differentially affect RNA editing between life cycle stages of Trypanosoma brucei
McDermott S M, et al.
Molecular and Cellular Biology, MCB-00790 (2015)
Identification by random mutagenesis of functional domains in KREPB5 that differentially affect RNA editing between life cycle stages of Trypanosoma brucei
McDermott S M, et al.
Molecular and cellular biology, MCB-00790 (2015)
Shuhan Chen et al.
Stem cell research, 45, 101804-101804 (2020-04-28)
Mutations in the Leucine rich repeat kinase 2 (LRRK2) gene are found in both familial and sporadic Parkinson's disease (PD), and are also associated with immune-related disorders including Crohn's disease (CD) and leprosy. We have generated two homozygous LRRK2 knockout
Meng Zhang et al.
Stem cell research, 41, 101602-101602 (2019-11-08)
Loss of function mutations in PARK2 (encoding PARKIN) cause autosomal recessive Parkinson's disease (PD), which often manifests at a juvenile age. Molecular and biochemical studies show that PARKIN functions as an E3 ubiquitin ligase controlling mitochondrial homeostasis. Yet, the exact
Huiling Wu et al.
The Journal of clinical investigation, 117(10), 2847-2859 (2007-09-15)
Ischemia/reperfusion injury (IRI) may activate innate immunity through the engagement of TLRs by endogenous ligands. TLR4 expressed within the kidney is a potential mediator of innate activation and inflammation. Using a mouse model of kidney IRI, we demonstrated a significant

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