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Merck
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67520

Sigma-Aldrich

1-Methyl-L-histidine

≥98.0% (TLC), for peptide synthesis

Sinonimo/i:

3-(1-Methylimidazol-4-yl)-L-alanine, 3-Methyl-L-histidine (archaic), Nτ-Methyl-L-histidine

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About This Item

Formula empirica (notazione di Hill):
C7H11N3O2
Numero CAS:
332-80-9
Peso molecolare:
169.18
Beilstein:
9727
Numero CE:
206-368-0
Numero MDL:
MFCD00005295
Codice UNSPSC:
12352209
ID PubChem:
57652090
NACRES:
NA.22

product name

1-Methyl-L-histidine, ≥98.0% (TLC)

Saggio

≥98.0% (TLC)

Forma fisica

powder

Attività ottica

[α]20/D −24±1°, c = 2% in H2O

Impiego in reazioni chimiche

reaction type: solution phase peptide synthesis

Impurezze

≤5% water

Punto di fusione

~240 °C (dec.) (lit.)

applicazioni

peptide synthesis

Stringa SMILE

Cn1cnc(C[C@H](N)C(O)=O)c1

InChI

1S/C7H11N3O2/c1-10-3-5(9-4-10)2-6(8)7(11)12/h3-4,6H,2,8H2,1H3,(H,11,12)/t6-/m0/s1
BRMWTNUJHUMWMS-LURJTMIESA-N

Categorie correlate

Confezionamento

Bottomless glass bottle. Contents are inside inserted fused cone.

Altre note

Natural but non-proteinogenic amino acid; employed as index of muscle protein breakdown

Codice della classe di stoccaggio

11 - Combustible Solids

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

Eyeshields, Gloves, type N95 (US)

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3-Methylhistidine as a measure of skeletal muscle protein breakdown in human subjects: the case for its continued use.
F J Ballard et al.
Clinical science (London, England : 1979), 65(3), 209-215 (1983-09-01)
P C Colombani et al.
International journal of sport nutrition, 9(2), 181-201 (1999-06-11)
A field study was performed to investigate the acute influence of a milk protein hydrolysate supplemented drink (CHO+PRO) on metabolism during and after a marathon run compared to the same drink without protein (CHO). Carbohydrate metabolites and hormones were not
J Sjölin et al.
Metabolism: clinical and experimental, 38(1), 23-29 (1989-01-01)
The present investigation was undertaken in order to determine the release of 3-methylhistidine (3MH) from the splanchnic region and from the leg, and the contributions these make to the increase in urinary 3MH excretion in infection. Thirteen febrile patients with
J Sjölin et al.
Metabolism: clinical and experimental, 36(12), 1175-1184 (1987-12-01)
In order to investigate whether the urinary excretion of 1-methylhistidine (1MH) might serve as an objective indicator of meat ingestion and exogenous 3-methylhistidine (3MH) intake, healthy subjects were fed an ovolactovegetarian diet. At five-day intervals they were given meat of
J Sjölin et al.
The American journal of clinical nutrition, 50(6), 1407-1414 (1989-12-01)
This investigation was undertaken to determine the splanchnic exchange of 3-methylhistidine (3MH) in infection. Hepatic vein, femoral vein, and radial artery catheterizations were performed in 12 febrile patients with infections of varying severity. Differences in arteriovenous 3MH, analyzed by high-performance
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