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M3404

(±)-p-Methoxyamphetamin -hydrochlorid

Name: (±)-p-Methoxyamphetamin -hydrochlorid
Brand: SIGMA

Synonym(e):

PMA -hydrochlorid

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About This Item

Empirische Formel (Hill-System):

C₁₀H₁₅NO · HCl

CAS-Nummer:

52740-56-4

Molekulargewicht:

201.69

MDL-Nummer:

MFCD00051431

UNSPSC-Code:

12352200

PubChem Substanz-ID:

24896840

Arzneimittelkontrolle

USDEA Schedule I; Home Office Schedule 1; stupéfiant (France); kontrollierte Droge in Deutschland; regulated under CDSA - not available from Sigma-Aldrich Canada

Methode(n)

HPLC: suitable
gas chromatography (GC): suitable

Anwendung(en)

forensics and toxicology
veterinary

SMILES String

Cl.COc1ccc(CC(C)N)cc1

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Biochem./physiol. Wirkung

CNS stimulant and hallucinogen; more potent transport inhibitor and releaser of serotonin than of dopamine.

Piktogramme

GHS06

Signalwort

Danger

H-Sätze

H301

P-Sätze

P301 + P310

Gefahreneinstufungen

Acute Tox. 3 Oral

Lagerklassenschlüssel

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

WGK

WGK 3

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

dust mask type N95 (US), Eyeshields, Faceshields, Gloves

Hier finden Sie alle aktuellen Versionen:

Analysenzertifikate (COA)

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Suche nach COA

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4-Ethoxyamphetamine: effects on intracranial self-stimulation and in vitro uptake and release of 3H-dopamine and 3H-serotonin in the brains of rats.

K M Hegadoren et al.

Journal of psychiatry & neuroscience : JPN, 19(1), 57-62 (1994-01-01)

Experiments were conducted to compare the effects of 4-ethoxyamphetamine, a novel "designer" amphetamine, with (+)-amphetamine and an earlier "designer" amphetamine, 4-methoxyamphetamine, on rats. (+)-Amphetamine significantly decreased frequency threshold measures in an intracranial self-stimulation (ICSS) procedure using medial forebrain bundle electrodes

Interactions of amphetamine analogs with human liver CYP2D6.

D Wu et al.

Biochemical pharmacology, 53(11), 1605-1612 (1997-06-01)

The interaction of fifteen amphetamine analogs with the genetically polymorphic enzyme CYP2D6 was examined. All fourteen phenylisopropylamines tested were competitive inhibitors of CYP2D6 in human liver microsomes. The presence of a methylenedioxy group in the 3,4-positions of both amphetamine (Ki

In vivo analysis of serotonin clearance in rat hippocampus reveals that repeated administration of p-methoxyamphetamine (PMA), but not 3,4-methylenedioxymethamphetamine (MDMA), leads to long-lasting deficits in serotonin transporter function.

Paul D Callaghan et al.

Journal of neurochemistry, 100(3), 617-627 (2006-12-22)

p-Methoxyamphetamine (PMA) has been implicated in fatalities as a result of 'ecstasy' (MDMA) overdose worldwide. Like MDMA, acute effects are associated with marked changes in serotonergic neurotransmission, but the long-term effects of PMA are poorly understood. The aim of this

Fatal paramethoxy-amphetamine (PMA) poisoning in the Australian Capital Territory.

Paul G Lamberth et al.

The Medical journal of Australia, 188(7), 426-426 (2008-04-09)

The effect of amphetamine analogs on cleaved microtubule-associated protein-tau formation in the rat brain.

M M W Straiko et al.

Neuroscience, 144(1), 223-231 (2006-11-07)

The present study quantified the cleaved form of the microtubule-associated protein tau (cleaved MAP-tau, C-tau), a previously demonstrated marker of CNS toxicity, following the administration of monoamine-depleting regimens of the psychostimulant drugs amphetamine (AMPH), methamphetamine (METH), +/-3,4-methylenedioxymethamphetamine (MDMA), or para-methoxyamphetamine

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