RAB1162
Rat Renin-1 ELISA Kit
for cell culture supernatants, plasma, and serum samples
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1 KIT
R$ 3.775,00
R$ 3.775,00
Previsão de entrega em02 de junho de 2025
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1 KIT
R$ 3.775,00
About This Item
Código UNSPSC:
41116158
NACRES:
NA.32
R$ 3.775,00
Previsão de entrega em02 de junho de 2025
Produtos recomendados
reatividade de espécies
rat
técnica(s)
ELISA: suitable
assay range
inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 68 pg/ml
Condições de expedição
wet ice
temperatura de armazenamento
−20°C
Categorias relacionadas
Descrição geral
This ELISA antibody pair detects Rat Renin (REN)
Aplicação
For research use only. Not for use in diagnostic procedures.
Please refer to the attached Protocolfor details.
Please refer to the attached Protocolfor details.
Outras notas
A sample Certificate of Analysis is available for this product. Please type the word sample in the text box provided for lot number.
Palavra indicadora
Warning
Frases de perigo
Declarações de precaução
Classificações de perigo
Met. Corr. 1
Código de classe de armazenamento
8A - Combustible corrosive hazardous materials
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
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Wolfram syndrome (WS) 1 is a rare monogenic neurodegenerative disorder caused by mutations in the gene encoding WFS1. Knowledge of the pathophysiology of WS is incomplete and to date, there is no treatment available. Here, we describe early deviations in
J Oh et al.
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Myeloid cells are known mediators of hypertension, but their role in initiating renin-induced hypertension has not been studied. Vitamin D deficiency causes pro-inflammatory macrophage infiltration in metabolic tissues and is linked to renin-mediated hypertension. We tested the hypothesis that impaired
Junda Hu et al.
Acta physiologica (Oxford, England), 232(1), e13612-e13612 (2020-12-31)
The use of calcineurin inhibitors such as cyclosporine A (CsA) for immunosuppression after solid organ transplantation is commonly limited by renal side effects. CsA-induced deterioration of glomerular filtration rate and sodium retention may be related to juxtaglomerular dysregulation as a
Dong Cheng et al.
Molecular medicine reports, 22(5), 4151-4162 (2020-10-02)
Clinical application of doxorubicin (DOX) is hampered by its potential cardiotoxicity, however angiotensin receptor blockers could attenuate DOX‑induced cardiomyopathy. The present study tested the hypothesis that simultaneous administration of valsartan (Val) with DOX could prevent DOX‑induced myocardial injury by modulating myocardial NAD(P)H
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