GF73191955
Iron
foil, light tested, 50x50mm, thickness 0.015mm, 99.85%
Sinônimo(s):
Iron, FE000170
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About This Item
Produtos recomendados
Ensaio
99.85%
forma
foil
fabricante/nome comercial
Goodfellow 731-919-55
resistividade
9.71 μΩ-cm
tamanho × espessura
50x50 mm × 0.015 mm
pb
2750 °C (lit.)
pf
1535 °C (lit.)
densidade
7.86 g/mL at 25 °C (lit.)
cadeia de caracteres SMILES
[Fe]
InChI
1S/Fe
chave InChI
XEEYBQQBJWHFJM-UHFFFAOYSA-N
Descrição geral
For updated SDS information please visit www.goodfellow.com.
Informações legais
Product of Goodfellow
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Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
Water science and technology : a journal of the International Association on Water Pollution Research, 59(12), 2503-2507 (2009-06-23)
Zero valent iron has been successfully used for the degradation of a wide range of contaminants. However, this reaction of using ZVI particle produces a large quantity of iron sludge. To solve the problem, we report the synthesis of self-organized
50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Proceedings of the National Academy of Sciences of the United States of America, 111(14), 5171-5176 (2014-04-08)
The enzymes PhnY and PhnZ comprise an oxidative catabolic pathway that enables marine bacteria to use 2-aminoethylphosphonic acid as a source of inorganic phosphate. PhnZ is notable for catalyzing the oxidative cleavage of a carbon-phosphorus bond using Fe(II) and dioxygen
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
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