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46102

Supelco

Fludioxonil

PESTANAL®, analytical standard

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About This Item

Empirische Formel (Hill-System):
C12H6F2N2O2
CAS-Nummer:
131341-86-1
Molekulargewicht:
248.19
Beilstein:
8393936
MDL-Nummer:
MFCD01631158
UNSPSC-Code:
41116107
PubChem Substanz-ID:
329756604
NACRES:
NA.24

Qualität

analytical standard

Produktlinie

PESTANAL®

Haltbarkeit

limited shelf life, expiry date on the label

Anwendung(en)

agriculture
environmental

Format

neat

SMILES String

FC1(F)Oc2cccc(c2O1)-c3c[nH]cc3C#N

InChI

1S/C12H6F2N2O2/c13-12(14)17-10-3-1-2-8(11(10)18-12)9-6-16-5-7(9)4-15/h1-3,5-6,16H

InChIKey

MUJOIMFVNIBMKC-UHFFFAOYSA-N

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Verwandte Kategorien

Anwendung

  • Fludioxonil fungicide in agricultural biotech: Fludioxonil′s effectiveness against the two-component histidine kinase Bos1 in Botrytis cinerea was explored, revealing its binding mode and molecular mechanism. This research underscores its critical role as a fungicide in agricultural biotechnology, providing insights that could lead to improved crop protection strategies (Yin et al., 2024).
  • Fludioxonil biochemical research for pathogen resistance: The study on Fusarium species causing soybean root rot examined the genetic basis of differing sensitivities to DMI fungicides, including fludioxonil. This contributes to a deeper understanding of fungal resistance mechanisms, enhancing the development of more effective fungicidal treatments (Zhang et al., 2024).
  • Fludioxonil antifungal properties in plant disease control: Research characterized fludioxonil and phenamacril dual resistant mutants of Fusarium graminearum, highlighting fludioxonil′s critical role in managing resistance in pathogens and its ongoing relevance in managing crop diseases (Wen et al., 2024).

Rechtliche Hinweise

PESTANAL is a registered trademark of Merck KGaA, Darmstadt, Germany

Piktogramme

Environment
GHS09

Signalwort

Warning

H-Sätze

H410

Gefahreneinstufungen

Aquatic Acute 1 - Aquatic Chronic 1

Lagerklassenschlüssel

11 - Combustible Solids

WGK

WGK 2

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

Eyeshields, Gloves

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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Paolo Tremolada et al.
Bulletin of environmental contamination and toxicology, 85(3), 229-234 (2010-07-27)
A first field study was conducted to investigate the possible adverse effects that seeds dressed with neonicotinoid insecticides pose to honeybees during sowing. It was observed that in the exposure hives bee mortality increased on the day of sowing and
Daisuke Hagiwara et al.
Fungal genetics and biology : FG & B, 46(11), 868-878 (2009-07-15)
In filamentous fungi, the His-Asp phosphorelay signaling system and HOG pathway are involved in the action of the fungicides, fludioxonil, and iprodione, as well as osmotic and oxidative stress responses. Aspergillusnidulans response regulators (RRs), SskA and SrrA, and histidine kinase
Roger R Lew
Fungal genetics and biology : FG & B, 47(8), 721-726 (2010-06-16)
The internal hydrostatic pressure (turgor) of the filamentous fungus Neurospora crassa is regulated at about 400-500 kiloPascals, primarily by an osmotic MAP kinase cascade which activates ion uptake from the extracellular medium and glycerol synthesis. In the absence of hyperosmotic
Allison McCormick et al.
PloS one, 7(6), e38262-e38262 (2012-06-08)
Two-component signaling systems are widespread in bacteria, but also found in fungi. In this study, we have characterized TcsC, the only Group III two-component sensor kinase of Aspergillus fumigatus. TcsC is required for growth under hyperosmotic stress, but dispensable for
Kentaro Furukawa et al.
FEBS letters, 586(16), 2417-2422 (2012-06-13)
The fungicide fludioxonil is used to control plant-pathogenic fungi by causing improper activation of the Hog1-type MAPK. However, the appearance of fludioxonil resistant mutants, mostly caused by mutations in the group III histidine kinases, poses a serious problem. Moreover, such
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