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S0882

Sigma-Aldrich

Sodium selenate

≥95% (elemental analysis)

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10 G
$84.87
100 G
$265.00

About This Item

Linear Formula:
Na2SeO4
CAS Number:
13410-01-0
Molecular Weight:
188.94
EC Number:
236-501-8
MDL number:
MFCD00003490
UNSPSC Code:
12352302
PubChem Substance ID:
329824181
NACRES:
NA.55
assay:
≥95% (elemental analysis)
form:
powder or crystals

$84.87

List Price$94.30Save 10%
Web-Only Promotion

Available to ship onMay 01, 2025Details

Request a Bulk Order

Quality Level

200

assay

≥95% (elemental analysis)

form

powder or crystals

pH

5.5-7.5 (25 °C, 18.9 g/L)

SMILES string

[Na+].[Na+].[O-][Se]([O-])(=O)=O

InChI

1S/2Na.H2O4Se/c;;1-5(2,3)4/h;;(H2,1,2,3,4)/q2*+1;/p-2

InChI key

MHQOTKLEMKRJIR-UHFFFAOYSA-L

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Application


  • Selenium nanoparticles alleviate ischemia reperfusion injury-induced acute kidney injury by modulating GPx-1/NLRP3/Caspase-1 pathway.: This study demonstrates that selenium nanoparticles, including sodium selenate, mitigate acute kidney injury caused by ischemia reperfusion. The nanoparticles modulate key pathways, offering potential therapeutic applications in renal injury treatment (Wang et al., 2022).

  • Phosphoproteomic profiling of selenate-treated Alzheimer′s disease model cells.: This research provides a detailed phosphoproteomic profile of Alzheimer′s disease model cells treated with sodium selenate, uncovering its effects on protein phosphorylation and implications for neuroprotection (Chen et al., 2014).

signalword

Danger

Hazard Classifications

Acute Tox. 2 Inhalation - Acute Tox. 2 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Skin Irrit. 2 - STOT RE 2

Storage Class

6.1B - Non-combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
BCCL4484
BCCK3757
BCCH0626
BCCH7647
BCCF0410

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Janet van Eersel et al.
Proceedings of the National Academy of Sciences of the United States of America, 107(31), 13888-13893 (2010-07-21)
Alzheimer's disease (AD) brains are characterized by amyloid-beta-containing plaques and hyperphosphorylated tau-containing neurofibrillary tangles (NFTs); however, in frontotemporal dementia, the tau pathology manifests in the absence of overt amyloid-beta plaques. Therapeutic strategies so far have primarily been targeting amyloid-beta, although
J A Hall et al.
Journal of animal science, 90(2), 568-576 (2011-10-04)
Although the essentiality of dietary Se for sheep has been known for decades, the chemical source and Se dosage for optimal health remain unclear. In the United States, the Food and Drug Administration (FDA) regulates Se supplementation, regardless of the
N M Corcoran et al.
British journal of cancer, 103(4), 462-468 (2010-07-22)
Angiogenesis is fundamental to the progression of many solid tumours including prostate cancer. Sodium selenate is a small, water-soluble, orally bioavailable activator of PP2A phosphatase with anti-angiogenic properties. This was a dose-escalation phase I study in men with asymptomatic, chemotherapy-naïve
Nigel C Jones et al.
Neurobiology of disease, 45(3), 897-901 (2011-12-21)
Tau hyperphosphorylation has been implicated in the pathogenesis of a variety of forms of human epilepsy. Here we investigated whether treatment with sodium selenate, a drug which reduces pathological hyperphosphorylated tau by enhancement of PP2A activity, would inhibit seizures in
Beatrice Pezzarossa et al.
Journal of the science of food and agriculture, 92(4), 781-786 (2011-09-29)
We investigated the effects of the foliar and fruit application of sodium selenate on selenium (Se) accumulation, fruit growth and ripening in peach and pear. Trials were conducted in two growing seasons. In 2008 selenate was applied at a rate
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