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T1162

Sigma-Aldrich

Tetramethylrhodamine isothiocyanate–Dextran

average mol wt 65,000-85,000

Synonym(s):

TRITC-Dextran

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About This Item

MDL number:
MFCD00132418
UNSPSC Code:
12352201
NACRES:
NA.25

form

powder

mol wt

average mol wt 65,000-85,000

extent of labeling

~1 TRITC per 100 glucose residues

color

purple to pink

storage temp.

2-8°C

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General description

Tetramethylrhodamine isothiocyanate-Dextran/TRITC-Dextran is a fluorochrome.

Application

Tetramethylrhodamine isothiocyanate-Dextran has been used to evaluate the role of pores (and channels) in the initial stages of amylolysis. It has also been used in protein-fluorescent conjugation.

Other Notes

To gain a comprehensive understanding of our extensive range of Polysaccharides for your research, we encourage you to visit our Carbohydrates Category page.

pictograms

Exclamation mark
GHS07

signalword

Warning

Hazard Classifications

Acute Tox. 4 Dermal - Acute Tox. 4 Inhalation - Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

ppe

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

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Amylase binding to starch granules under hydrolysing and non-hydrolysing conditions
Dhital S, et al.
Carbohydrate Polymers, 113, 97-107 (2014)
Evaluation of fluorescent plasma markers for in vivo microscopy of the microcirculation
Reeves KJ, et al.
Journal of Vascular Research, 49(2), 132-143 (2012)
Nicholas E Propson et al.
The Journal of clinical investigation, 131(1) (2020-09-30)
Dysfunction of immune and vascular systems has been implicated in aging and Alzheimer disease; however, their interrelatedness remains poorly understood. The complement pathway is a well-established regulator of innate immunity in the brain. Here, we report robust age-dependent increases in
Hematopoietic Cancers
Immune Response, 1025-1063 (2006)
Stephen Sakuma et al.
Cancer discovery, 11(1), 176-193 (2020-09-30)
Nuclear pore complexes (NPC) are the central mediators of nucleocytoplasmic transport. Increasing evidence shows that many cancer cells have increased numbers of NPCs and become addicted to the nuclear transport machinery. How reducing NPC numbers affects the physiology of normal
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