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Merck
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重要文件

V800129

Sigma-Aldrich

氯化铜(II)

LR, ≥98%

同義詞:

氯化铜

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About This Item

線性公式:
CuCl2
CAS號碼:
分子量::
134.45
EC號碼:
MDL號碼:
分類程式碼代碼:
12352302
PubChem物質ID:
暫時無法取得訂價和供貨情況

等級

LR

產品線

Vetec

化驗

≥98%

形狀

powder

mp

620 °C (lit.)

密度

3.386 g/mL at 25 °C (lit.)

SMILES 字串

Cl[Cu]Cl

InChI

1S/2ClH.Cu/h2*1H;/q;;+2/p-2

InChI 密鑰

ORTQZVOHEJQUHG-UHFFFAOYSA-L

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法律資訊

Vetec is a trademark of Merck KGaA, Darmstadt, Germany

訊號詞

Danger

危險分類

Acute Tox. 4 Dermal - Acute Tox. 4 Oral - Aquatic Acute 1 - Aquatic Chronic 2 - Eye Dam. 1 - Skin Irrit. 2

儲存類別代碼

8A - Combustible corrosive hazardous materials

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Kenji Funaki et al.
Organic letters, 14(24), 6186-6189 (2012-12-12)
Direct arylation of thiophenes and benzothiophenes with aryltrimethylsilanes was effectively catalyzed by PdCl(2)(MeCN)(2) in the presence of CuCl(2) as an oxidant. The reaction preferentially occurred at the β-position of both thiophenes and benzothiophenes.
Mónica João Barros Amorim et al.
Environmental pollution (Barking, Essex : 1987), 164, 164-168 (2012-03-01)
Environmental effects of copper nanoparticles are little studied in terrestrial ecosystems. In the present article, the toxicity of copper nanoparticles (Cu-NP) on the enchytraeid Enchytraeus albidus is compared to the toxicity of a copper-salt (CuCl(2)). The effect parameters studied were
Iron(II)-catalyzed trifluoromethylation of potassium vinyltrifluoroborates.
Andrew T Parsons et al.
Angewandte Chemie (International ed. in English), 51(12), 2947-2950 (2012-02-14)
Francesca Cima et al.
Chemosphere, 89(1), 19-29 (2012-05-01)
After the widespread ban of TBT, due to its severe impact on coastal biocoenoses, mainly related to its immunosuppressive effects on both invertebrates and vertebrates, alternative biocides such as Cu(I) salts and the triazine Irgarol 1051, the latter previously used
Pieter Spincemaille et al.
Toxicology and applied pharmacology, 280(2), 345-351 (2014-08-20)
Wilson disease (WD) is caused by accumulation of excess copper (Cu) due to a mutation in the gene encoding the liver Cu transporter ATP7B, and is characterized by acute liver failure or cirrhosis and neuronal cell death. We investigated the

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