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About This Item
線性公式:
CH3(CH2)7NHC(=NH)NH2 · 1/2H2SO4
CAS號碼:
分子量：:
220.32
MDL號碼:
分類程式碼代碼:
12352100
PubChem物質ID:
NACRES:
NA.22
推薦產品
化驗
97%
SMILES 字串
OS(O)(=O)=O.CCCCCCCCNC(N)=N.CCCCCCCCNC(N)=N
InChI
1S/2C9H21N3.H2O4S/c2*1-2-3-4-5-6-7-8-12-9(10)11;1-5(2,3)4/h2*2-8H2,1H3,(H4,10,11,12);(H2,1,2,3,4)
InChI 密鑰
DTTVLDRWEFQROK-UHFFFAOYSA-N
一般說明
1-Octylguanidine hemisulfate is a guanidine derivative. Octyl guanidine sulfate is prepared from S-methylisothiourea sulfate and octylamine.
訊號詞
Warning
危險聲明
危險分類
Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
標靶器官
Respiratory system
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
dust mask type N95 (US), Eyeshields, Gloves
J R de Weille et al.
Brain research, 445(1), 1-11 (1988-03-29)
Kinetics of pyrethroid-modified sodium channels and the interaction of N-octylguanidine with the modified channels have been studied with internally perfused and voltage-clamped squid giant axons. The pyrethroids used were 1R-cis-phenothrin; 1R-cis-permethrin; 1R-cis-cyphenothrin; and 1R-cis-deltamethrin. Modification of sodium channels by pyrethroids
Studies on nitrogen metabolism in tobacco plants IX. Effect of various compounds on proline biosynthesis in the green leaves.
Noguchi M, et al.
Plant & Cell Physiology, 9(1), 35-47 (1968)
Victoriano Pérez-Vázquez et al.
Journal of bioenergetics and biomembranes, 35(3), 231-241 (2003-09-19)
The yeast mitochondrial unspecific channel (YMUC) sensitivity to inorganic (Ca2+ or Mg2+) or organic (hexyl or octyl-guanidine) cations was measured. The rate of oxygen consumption in State 3 and State 4, the transmembrane potential (deltapsi), mitochondrial swelling, and the polyethylene-glycol
Natalia Pavón et al.
Journal of biochemistry, 149(2), 211-217 (2010-11-30)
Mercurials are known to induce morphological and functional modifications in kidney. The protective effect of octylguanidine on the injury induced by Hg(2+) on renal functions was studied. Octylguanidine administered at a dose of 10 mg/kg body weight prevented the damage
Natalia Pavón et al.
Endocrine, 35(2), 158-165 (2009-01-27)
Hyperthyroidism sensitizes the heart for reperfusion injury. As known, mitochondrial permeability transition underlies reperfusion heart damage. This study was undertaken to explore the protective effect of octylguanidine (OG), an inhibitor of permeability transition, on hearts from hyperthyroid rats subjected to
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