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Merck

SRE0012

Sigma-Aldrich

载脂蛋白 A-I 人

histidine-tagged, recombinant, expressed in HEK 293 cells, ≥98% (SDS-PAGE), lyophilized powder

别名:

APOA1, Apo-AI, C117399, MGC117399, 载脂蛋白 A-I

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About This Item

分類程式碼代碼:
12352202
NACRES:
NA.26

生物源

human

品質等級

重組細胞

expressed in HEK 293 cells

化驗

≥98% (SDS-PAGE)

形狀

lyophilized powder

UniProt登錄號

儲存溫度

−20°C

基因資訊

human ... APOA1(335)

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Ce produit, destiné à la recherche scientifique, est soumis à une réglementation spécifique en France, y compris pour les activités d′importation et d′exportation (Article L 1211-1 alinéa 2 du Code de la Santé Publique). L′acheteur (c′est-à-dire l′utilisateur final) est tenu d′obtenir une autorisation d′importation auprès du Ministère français de la Recherche, mentionné à l′article L1245-5-1 II du Code de la Santé Publique. En commandant ce produit, vous confirmez détenir l′autorisation d′importation requise.

RESEARCH USE ONLY. This product is regulated in France when intended to be used for scientific purposes, including for import and export activities (Article L 1211-1 paragraph 2 of the Public Health Code). The purchaser (i.e. enduser) is required to obtain an import authorization from the France Ministry of Research referred in the Article L1245-5-1 II. of Public Health Code. By ordering this product, you are confirming that you have obtained the proper import authorization.

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Nathalie Niyonzima et al.
Journal of immunology (Baltimore, Md. : 1950), 195(1), 257-264 (2015-05-31)
Chronic inflammation of the arterial wall is a key element in the development of atherosclerosis, and cholesterol crystals (CC) that accumulate in plaques are associated with initiation and progression of the disease. We recently revealed a link between the complement
Judit Cubedo et al.
Journal of lipid research, 56(9), 1762-1773 (2015-07-15)
Diabetic (DM) patients have exacerbated atherosclerosis and high CVD burden. Changes in lipid metabolism, lipoprotein structure, and dysfunctional HDL are characteristics of diabetes. Our aim was to investigate whether serum ApoA-I, the main protein in HDL, was biochemically modified in

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