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Merck

PLA0161

Sigma-Aldrich

Rabbit anti-MOF/MYST1 Antibody, Affinity Purified

Powered by Bethyl Laboratories, Inc.

别名:

FLJ14040, K(lysine) acetyltransferase 8, Lysine acetyltransferase 8, MOF, MOZ, MYST histone acetyltransferase 1, MYST-1, MYST1, SAS2 and TIP60 protein 1, YBF2/SAS3, ZC2HC8, hMOF, histone acetyltransferase MYST1, ortholog of Drosophila males absent on the first (MOF), probable histone acetyltransferase MYST1

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About This Item

分類程式碼代碼:
12352203
NACRES:
NA.41

生物源

rabbit

品質等級

抗體表格

affinity purified immunoglobulin

抗體產品種類

primary antibodies

等級

Powered by Bethyl Laboratories, Inc.

物種活性

human, mouse

技術

immunohistochemistry: 1:500- 1:2,000
immunoprecipitation (IP): 2-5 μg/mg
western blot: 1:1,000- 1:10,000

登錄號

BAB13924.1

運輸包裝

wet ice

儲存溫度

2-8°C

目標翻譯後修改

unmodified

基因資訊

免疫原

The epitope recognized by PLA0161 maps to a region between residue 1 and 50 of human males absent on the first (MYST histone acetyltransferase 1) using the numbering given in entry BAB13924.1 (GeneID 84148).

外觀

Tris-citrate/phosphate buffer, pH 7 to 8 containing 0.09% Sodium Azide

其他說明

MOF is a member of the MYST family of histone acetyltransferases (HAT). MOF has been shown to be required for histone H4 lysine K16-specific acetylation. MOF interacts with ATM (ataxia-telangiectasia-mutated) and may participate in the activation of ATM in response to DNA damage.

免責聲明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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儲存類別代碼

12 - Non Combustible Liquids

水污染物質分類(WGK)

nwg

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Mzwanele Ngubo et al.
Aging cell, 23(7), e14150-e14150 (2024-04-05)
Hutchinson-Gilford Progeria syndrome (HGPS) is a lethal premature aging disorder caused by a de novo heterozygous mutation that leads to the accumulation of a splicing isoform of Lamin A termed progerin. Progerin expression deregulates the organization of the nuclear lamina

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