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等級
natural
法律遵循
FDA 21 CFR 117
形狀
liquid
濃度
45.00-55.00% (titration by hydroxylamine)
50 wt. % in ethanol
折射率
n20/D 1.39 (lit.)
密度
0.868 g/mL at 20 °C
0.784 g/mL at 25 °C (lit.)
應用
flavors and fragrances
感官的
ethereal
儲存溫度
2-8°C
SMILES 字串
[H]C(C)=O
InChI
1S/C2H4O/c1-2-3/h2H,1H3
InChI 密鑰
IKHGUXGNUITLKF-UHFFFAOYSA-N
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訊號詞
Danger
危險分類
Carc. 1B - Eye Irrit. 2 - Flam. Liq. 2 - Muta. 2 - STOT SE 3
標靶器官
Respiratory system
儲存類別代碼
3 - Flammable liquids
水污染物質分類（WGK）
WGK 3
閃點（°F）
30.2 °F - closed cup
閃點（°C）
-1 °C - closed cup
其他客户在看
Hyo-Jung Kwon et al.
Hepatology (Baltimore, Md.), 60(1), 146-157 (2014-02-05)
Aldehyde dehydrogenase 2 (ALDH2) is the major enzyme that metabolizes acetaldehyde produced from alcohol metabolism. Approximately 40-50% of East Asians carry an inactive ALDH2 gene and exhibit acetaldehyde accumulation after alcohol consumption. However, the role of ALDH2 deficiency in the
Hsu-Sheng Yu et al.
Chemico-biological interactions, 188(3), 367-375 (2010-09-04)
Epidemiological studies have identified chronic alcohol consumption as a significant risk factor for cancers of the upper aerodigestive tract, including the oral cavity, pharynx, larynx and esophagus, and for cancer of the liver. Ingested ethanol is mainly oxidized by the
Mashiko Setshedi et al.
Oxidative medicine and cellular longevity, 3(3), 178-185 (2010-08-19)
Chronic alcohol abuse causes liver disease that progresses from simple steatosis through stages of steatohepatitis, fibrosis, cirrhosis, and eventually hepatic failure. In addition, chronic alcoholic liver disease (ALD), with or without cirrhosis, increases risk for hepatocellular carcinoma (HCC). Acetaldehyde, a
Tommaso Mello et al.
Molecular aspects of medicine, 29(1-2), 17-21 (2008-01-01)
Alcohol abuse is one of the major causes of liver fibrosis worldwide. Although the pathogenesis of liver fibrosis is a very complex phenomenon involving different molecular and biological mechanisms, several lines of evidence established that the first ethanol metabolite, acetaldehyde
Mikko Salaspuro
Journal of digestive diseases, 12(2), 51-59 (2011-03-16)
Aldehyde dehydrogenase (ALDH2) and alcohol dehydrogenase (ADH) gene polymorphisms associating with enhanced acetaldehyde exposure and markedly increased cancer risk in alcohol drinkers provide undisputable evidence for acetaldehyde being a local carcinogen not only in esophageal but also in gastric cancer.
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