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추천 제품
설명
Merck USA index - 14, 8468
Quality Level
분석
≥95% (SDS-PAGE)
양식
lyophilized
제조업체/상표
Calbiochem®
저장 조건
OK to freeze
기술
ELISA: suitable
solubility
water: 20 mg/mL
배송 상태
ambient
저장 온도
−20°C
일반 설명
Plasma protein that is useful as a stabilizing agent and as a carrier protein.
애플리케이션
Albumin, mouse has been used as a component of PBS for in vivo examination of (E, E)-1-fluoro-2,5-bis(3-hydroxycarbonyl-4-hydroxy)styrylbenzene (FSB) and yellow fluorescent protein (YFP) positive cells in mice.[1]
경고
Toxicity: Standard Handling (A)
재구성
Following reconstitution, aliquot and freeze (-20°C). Stock solutions are stable for up to 6 months at -20°C.
법적 정보
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
시험 성적서(COA)
제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.
이미 열람한 고객
Christian Schön et al.
PloS one, 7(12), e53547-e53547 (2013-01-10)
Tauopathies are widespread neurodegenerative disorders characterised by the intracellular accumulation of hyperphosphorylated tau. Especially in Alzheimer's disease, pathological alterations in the retina are discussed as potential biomarkers to improve early diagnosis of the disease. Using mice expressing human mutant P301S
Arieke Suzanna Berendina Kampstra et al.
Annals of the rheumatic diseases, 78(7), 908-916 (2019-06-04)
Autoantibodies against post-translationally modified proteins (anti-modified protein antibodies or AMPAs) are a hallmark of rheumatoid arthritis (RA). A variety of classes of AMPAs against different modifications on proteins, such as citrullination, carbamylation and acetylation, have now been described in RA.
Petar Marinković et al.
Brain : a journal of neurology, 142(4), 1051-1062 (2019-03-09)
Pathological alterations of tau protein play a significant role in the emergence and progression of neurodegenerative disorders. Tauopathies are characterized by detachment of the tau protein from neuronal microtubules, and its subsequent aberrant hyperphosphorylation, aggregation and cellular distribution. The exact
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