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Merck

G4901

透射电子显微镜用滤线栅

grid size 300 mesh × 83 μm pitch, copper

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About This Item

分類程式碼代碼:
41100000
NACRES:
MA.01

材料

copper

描述

Regular grids with center mark and solid border

包裝

vial of 100 grids

直徑

3.05 mm

網格尺寸

300 mesh × 83 μm pitch

孔寬度

63 μm

開口區尺寸

55%

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應用

透射电镜用载网可固定组织薄片,用于:
  • 人神经母细胞瘤细胞系的受损线粒体和自噬研究
  • 大鼠组织标本的组织学检验
  • 小鼠成肌细胞系的形态分析


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T Martinovic et al.
Tissue & cell, 52, 92-100 (2018-06-03)
Type 2 diabetes is an endocrine disorder characterized with hyperglycemia, hyperinsulinemia and insulin resistance. Morphological changes in cell nuclei in diabetes were recently detected. The aim of this study was to compare electron microscopic features of lymphocyte nuclei in type
İsmail Seçkin et al.
Romanian journal of morphology and embryology = Revue roumaine de morphologie et embryologie, 58(3), 823-830 (2017-12-19)
Puromycin aminonucleoside (PA) has been generally utilized as model of podocyte injury followed by massive proteinuria, severe damage on endocytotic activity of epithelial cells and postmodification of endocytosed compounds. However, total PA nephrosis (PAN) mechanism cannot be understood. We aimed
Shijun Yu et al.
Hepatology research : the official journal of the Japan Society of Hepatology, 49(11), 1329-1340 (2019-06-27)
B-cell lymphoma-2-associated transcription factor 1 (BCLAF1) is involved in various biological processes including tumorigenesis, but its function and expression in hepatocellular carcinoma (HCC) is little known, and its clinical value in HCC has not yet been defined. The protein level
Ismail Seckin et al.
Journal of biomedical science, 19, 24-24 (2012-02-18)
In experimentally induced puromycine aminonucleoside nephrosis (PAN) animal models, nephrotic syndrome with minimal change disease and focal and segmental sclerosis-like nephritis similar to that in human is demonstrated; however, the real mechanism of PAN is not yet elucidated. Platelet derived
Salime Pelin Ertürküner et al.
Pharmacological reports : PR, 66(2), 279-287 (2014-06-10)
Hyperglycemia and advanced glucose end substance (AGE) are responsible for excessive reactive oxygen species (ROS) production, which causes oxidative stress in diabetes mellitus. Oxidative stress and high blood pressure may cause injury and glomerulosclerosis in the kidney. End-stage kidney failure

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