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Merck

388866

Sigma-Aldrich

Brij® 93

average Mn ~357

别名:

聚氧乙烯(2)油醚, 聚氧乙烯月桂醚

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About This Item

线性分子式:
C18H35(OCH2CH2)nOH, n~2
CAS号:
分子量:
356.58
MDL號碼:
分類程式碼代碼:
12162002
NACRES:
NA.23

蒸汽壓力

0.26 psi ( 20 °C)

形狀

liquid

分子量

average Mn ~357

折射率

n20/D 1.462 (lit.)

密度

0.912 g/mL at 25 °C (lit.)

HLB

4

InChI

1S/C20H40O2/c1-2-3-4-5-6-7-8-9-10-11-12-13-14-15-16-17-19-22-20-18-21/h9-10,21H,2-8,11-20H2,1H3/b10-9-

InChI 密鑰

KWVPFECTOKLOBL-KTKRTIGZSA-N

一般說明

Brij® 93是一种非离子型表面活性剂,具有一个以由聚氧乙烯形式通过聚乙烯链分离的极性基团。其分子结构包括聚乙二醇(PEG)单链。其微溶于水且具有疏水性。

法律資訊

Brij is a registered trademark of Croda International PLC

象形圖

Exclamation markEnvironment

訊號詞

Warning

危險聲明

危險分類

Aquatic Chronic 2 - Skin Irrit. 2

儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 1

閃點(°F)

>464.0 °F - Equilibrium method

閃點(°C)

> 240 °C - Equilibrium method

個人防護裝備

Eyeshields, Gloves, type ABEK (EN14387) respirator filter


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Stabilization of High Ionic Strength Slurries Using Surfactant Mixtures: Molecular Factors That Determine Optimal Stability
Palla BJ, et al.
Journal of Colloid and Interface Science, 256(1), 143-152 (2002)
Optimization of a novel and improved thermosensitive liposome formulated with DPPC and a Brij surfactant using a robust in vitro system
Tagami T, et al.
J. Controlled Release, 154(3), 290-297 (2011)
Hydrothermal synthesis of nanocrystalline hydroxyapatite from phosphogypsum waste
Bensalah H, et al.
Journal of Environmental Chemical Engineering, 6(1), 1347-1352 (2018)
María Vergara-Barberán et al.
Journal of agricultural and food chemistry, 59(20), 10775-10780 (2011-09-13)
A method for the determination of fatty acids in vegetable oils by capillary electrophoresis with indirect UV-vis detection has been developed. The separation of fatty acids was optimized in terms of Brij surfactant nature and concentration and organic modifier (2-propanol)
Myriam Chentouf et al.
Journal of immunology (Baltimore, Md. : 1950), 179(1), 409-420 (2007-06-21)
The biological effects of rIgG(1) 13B8.2, directed against the CDR3-like loop on the D1 domain of CD4, are partly due to signals that prevent NF-kappaB nuclear translocation, but the precise mechanisms of action, particularly at the level of membrane proximal

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