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Sigma-Aldrich

meta-Phosphoric acid

ACS reagent, chips, 33.5-36.5%

Synonyme(s) :

Metaphosphoric acid

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About This Item

Formule linéaire :
(HPO3)n
Numéro CAS:
37267-86-0
Numéro MDL:
MFCD00011351
Code UNSPSC :
12352106
ID de substance PubChem :
329752524
Nomenclature NACRES :
NA.21

Qualité

ACS reagent

Forme

chips

Contient

57.0-63.0% sodium phosphite as stabilizer

Concentration

33.5-36.5%

Densité

2 g/cm3

Traces d'anions

MnO4- reducers: ≤0.02%
chloride (Cl-): ≤0.001%
nitrate (NO3-): ≤0.001%
sulfate (SO42-): ≤0.005%

Traces de cations

As: ≤1 ppm
Fe: ≤0.005%
heavy metals: ≤0.005% (by ICP)

Chaîne SMILES 

OP(=O)=O

InChI

1S/HO3P/c1-4(2)3/h(H,1,2,3)

Clé InChI

UEZVMMHDMIWARA-UHFFFAOYSA-N

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Catégories apparentées

Description générale

meta-Phosphoric acid is a polymer form of phosphoric acid. It is used as a catalyst as well as a reactant in chemical synthesis.

Application

meta-Phosphoric acid can be used as:      
  • A catalyst in combination with InCl3 to synthesize 1,8-dioxooctahydroxanthene derivatives by the reaction of cyclic β-diketones and aldehydes.      
  • A phosphate precursor to synthesize antimony phosphate (SbPO4) by reacting with antimony metal at high temperatures.

Pictogrammes

Corrosion
GHS05

Mention d'avertissement

Danger

Mentions de danger

H314

Classification des risques

Eye Dam. 1 - Skin Corr. 1

Code de la classe de stockage

8B - Non-combustible corrosive hazardous materials

Classe de danger pour l'eau (WGK)

WGK 3

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable

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Chand Basha Davuljigari et al.
International journal of molecular sciences, 22(11) (2021-07-03)
Acrylamide is a well characterized neurotoxicant known to cause neuropathy and encephalopathy in humans and experimental animals. To investigate the role of nuclear factor erythroid 2-related factor 2 (Nrf2) in acrylamide-induced neuropathy, male C57Bl/6JJcl adult mice were exposed to acrylamide
Sellamuthu S Gounder et al.
PloS one, 7(9), e45697-e45697 (2012-10-03)
Aging promotes accumulation of reactive oxygen/nitrogen species (ROS/RNS) in cardiomyocytes, which leads to contractile dysfunction and cardiac abnormalities. These changes may contribute to increased cardiovascular disease in the elderly. Inducible antioxidant pathways are regulated by nuclear erythroid 2 p45-related factor
Namakkal Soorappan Rajasekaran et al.
Antioxidants & redox signaling, 14(6), 957-971 (2010-12-04)
Inheritable missense mutations in small molecular weight heat-shock proteins (HSP) with chaperone-like properties promote self-oligomerization, protein aggregation, and pathologic states such as hypertrophic cardiomyopathy in humans. We recently described that human mutant αB-crystallin (hR120GCryAB) overexpression that caused protein aggregation cardiomyopathy
Alicia Madurga et al.
American journal of physiology. Lung cellular and molecular physiology, 306(7), L684-L697 (2014-02-11)
Arrested alveolarization is the pathological hallmark of bronchopulmonary dysplasia (BPD), a complication of premature birth. Here, the impact of systemic application of hydrogen sulfide (H2S) on postnatal alveolarization was assessed in a mouse BPD model. Exposure of newborn mice to
Vasanthi R Muthusamy et al.
Free radical biology & medicine, 52(2), 366-376 (2011-11-05)
Oxidative stress has been implicated in the pathogenesis of cardiovascular diseases, including myocardial hypertrophy and infarction. Although impairment of antioxidant defense mechanisms has been thought to provoke oxidative stress-induced myocardial dysfunction, it has been difficult to clearly demonstrate. Nuclear erythroid
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