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GF87455234

Iron

foil, 10mm disks, thickness 0.0125mm, 99.85%

Sinônimo(s):

Iron, FE000160

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About This Item

Fórmula linear:
Fe
Número CAS:
Peso molecular:
55.85
Número MDL:
Código UNSPSC:
12141721
ID de substância PubChem:
NACRES:
NA.23

Ensaio

≥99.85%

forma

foil

fabricante/nome comercial

Goodfellow 874-552-34

resistividade

9.71 μΩ-cm

pb

2750 °C (lit.)

pf

1535 °C (lit.)

densidade

7.86 g/mL at 25 °C (lit.)

cadeia de caracteres SMILES

[Fe]

InChI

1S/Fe

chave InChI

XEEYBQQBJWHFJM-UHFFFAOYSA-N

Descrição geral

For updated SDS information please visit www.goodfellow.com.

Informações legais

Product of Goodfellow

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Arnold L Demain et al.
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
Jun-Won Jang et al.
Water science and technology : a journal of the International Association on Water Pollution Research, 59(12), 2503-2507 (2009-06-23)
Zero valent iron has been successfully used for the degradation of a wide range of contaminants. However, this reaction of using ZVI particle produces a large quantity of iron sludge. To solve the problem, we report the synthesis of self-organized
50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
Pamela J Kling
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
Joseph D Mancias et al.
Nature, 509(7498), 105-109 (2014-04-04)
Autophagy, the process by which proteins and organelles are sequestered in double-membrane structures called autophagosomes and delivered to lysosomes for degradation, is critical in diseases such as cancer and neurodegeneration. Much of our understanding of this process has emerged from

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