GF69207240
Copper - O.F.H.C.
foil, 10m coil, thickness 0.075mm, half hard, 99.95+%
Sinônimo(s):
Copper, CV000540
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About This Item
Fórmula empírica (Notação de Hill):
Cu
Número CAS:
Peso molecular:
63.55
Número MDL:
Código UNSPSC:
12141711
ID de substância PubChem:
NACRES:
NA.23
Ensaio
99.95%
forma
foil
fabricante/nome comercial
Goodfellow 692-072-40
resistividade
1.673 μΩ-cm, 20°C
C × espessura
10 m × 0.075 mm
pb
2567 °C (lit.)
pf
1083.4 °C (lit.)
densidade
8.94 g/mL at 25 °C (lit.)
cadeia de caracteres SMILES
[Cu]
InChI
1S/Cu
chave InChI
RYGMFSIKBFXOCR-UHFFFAOYSA-N
Descrição geral
For updated SDS information please visit www.goodfellow.com.
Informações legais
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Julie E Gleason et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(16), 5866-5871 (2014-04-09)
The human fungal pathogens Candida albicans and Histoplasma capsulatum have been reported to protect against the oxidative burst of host innate immune cells using a family of extracellular proteins with similarity to Cu/Zn superoxide dismutase 1 (SOD1). We report here
Giulia Cheloni et al.
Aquatic toxicology (Amsterdam, Netherlands), 155, 275-282 (2014-07-30)
The present study showed the important role of light intensity and spectral composition on Cu uptake and effects on green alga Chlamydomonas reinhardtii. High-intenisty light (HL) increased cellular Cu concentrations, but mitigated the Cu-induced decrease in chlorophyll fluorescence, oxidative stress
Alina Fedoseienko et al.
Annals of the New York Academy of Sciences, 1314, 6-14 (2014-04-05)
Copper is an important cofactor in numerous biological processes in all living organisms. However, excessive copper can be extremely toxic, so it is vital that the copper level within a cell is tightly regulated. The damaging effect of copper is
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
Adam Southon et al.
Metallomics : integrated biometal science, 5(10), 1346-1356 (2013-08-02)
Copper (Cu) is an essential redox active metal that is potentially toxic in excess. Multicellular organisms acquire Cu from the diet and must regulate uptake, storage, distribution and export of Cu at both the cellular and organismal levels. Systemic Cu
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