GF19723136
Copper - O.F.H.C.
foil, 25mm disks, thickness 0.125mm, hard, 99.95+%
Sinônimo(s):
Copper, CV000590
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About This Item
Ensaio
99.95%
forma
foil
fabricante/nome comercial
Goodfellow 197-231-36
resistividade
1.673 μΩ-cm, 20°C
diâmetro × espessura
25 mm × 0.125 mm
pb
2567 °C (lit.)
pf
1083.4 °C (lit.)
densidade
8.94 g/mL at 25 °C (lit.)
cadeia de caracteres SMILES
[Cu]
InChI
1S/Cu
chave InChI
RYGMFSIKBFXOCR-UHFFFAOYSA-N
Categorias relacionadas
Descrição geral
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Informações legais
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Nature, 509(7501), 492-496 (2014-04-11)
The BRAF kinase is mutated, typically Val 600→Glu (V600E), to induce an active oncogenic state in a large fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide spectrum of other cancers. BRAF(V600E) phosphorylates and
Environmental pollution (Barking, Essex : 1987), 194, 78-85 (2014-08-06)
Copper (Cu) is known to accumulate in agricultural soils receiving urban waste products as fertilizers. We here report the use of the leucine incorporation technique to determine pollution-induced community tolerance (Leu-PICT) to Cu in a long-term agricultural field trial. A
Proceedings of the National Academy of Sciences of the United States of America, 111(16), 5866-5871 (2014-04-09)
The human fungal pathogens Candida albicans and Histoplasma capsulatum have been reported to protect against the oxidative burst of host innate immune cells using a family of extracellular proteins with similarity to Cu/Zn superoxide dismutase 1 (SOD1). We report here
Environmental pollution (Barking, Essex : 1987), 193, 205-215 (2014-07-25)
Intra-specific variability of root biomass production (RP) of six rooted macrophytes, i.e. Juncus effusus, Phragmites australis, Schoenoplectus lacustris, Typha latifolia, Phalaris arundinacea, and Iris pseudacorus grown from clones, in response to Cu exposure was investigated. Root biomass production varied widely
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
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