EM0200
Osteo-Bed Bone Embedding Kit
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About This Item
UNSPSC Code:
12171500
NACRES:
NA.47
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Productos recomendados
Application
Suitable for use with large and small mineralized (undecalcified) bone sections. Yields clear, hard blocks for cutting sections. Not water-soluble.
Recommended products
Osteo-Bed Bone Embedding Solvent (O8639) is available to remove plastic from sections.
Solo componentes del kit
Referencia del producto
Descripción
- Osteo-Bed Bone Embedding Resin A (O8514) 900 mL
- Osteo-Bed Bone Embedding Catalyst (O8764) 2 x 12
Optional
Referencia del producto
Descripción
Precios
signalword
Danger
Hazard Classifications
Aquatic Acute 1 - Aquatic Chronic 1 - Eye Irrit. 2 - Flam. Liq. 2 - Org. Perox. D - Repr. 1B - Skin Irrit. 2 - Skin Sens. 1 - STOT SE 3
target_organs
Respiratory system
Storage Class
5.2 - Organic peroxides and self-reacting hazardous materials
flash_point_f
50.0 °F
flash_point_c
10 °C
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Nur-Vaizura Mohamad et al.
International journal of medical sciences, 18(16), 3665-3673 (2021-11-19)
Menopause is the leading cause of osteoporosis for elderly women due to imbalanced bone remodelling in the absence of oestrogen. The ability of tocotrienol in reversing established bone loss due to oestrogen deficiency remains unclear despite the plenitude of evidence
Colin K Yee et al.
Clinical & experimental metastasis, 25(8), 903-911 (2008-09-25)
In the current study, we examine heparin's anti-metastatic properties by using a well-defined mouse model of osteolytic bone metastasis. C57BL/6 mice were treated with increasing doses of unfractionated heparin (15, 20, or 25 units/mouse) 30 min prior to the left
Lei Qin et al.
Journal of orthopaedic translation, 34, 60-72 (2022-05-27)
The key focal adhesion protein β1 integrin plays an essential role in early skeletal development. However, roles of β1 integrin expression in osteocytes during the regulation of bone homeostasis and mechanotransduction are incompletely understood. To study the in vivo function
Kindlin-2 mediates mechanotransduction in bone by regulating expression of Sclerostin in osteocytes.
Lei Qin et al.
Communications biology, 4(1), 402-402 (2021-03-27)
Osteocytes act as mechanosensors in bone; however, the underlying mechanism remains poorly understood. Here we report that deleting Kindlin-2 in osteocytes causes severe osteopenia and mechanical property defects in weight-bearing long bones, but not in non-weight-bearing calvariae. Kindlin-2 loss in
Qinnan Yan et al.
The Journal of biological chemistry, 298(3), 101639-101639 (2022-01-30)
Phosphatidylinositol-4-phosphate 5-kinase type-1 gamma (Pip5k1c) is a lipid kinase that plays a pivotal role in the regulation of receptor-mediated calcium signaling in multiple tissues; however, its role in the skeleton is not clear. Here, we show that while deleting Pip5k1c
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